Catalytically active Yersinia outer protein P induces cleavage of RIP and caspase-8 at the level of the DISC independently of death receptors in dendritic cells.
about
Cell death programs in Yersinia immunity and pathogenesisYersinia type III effectors perturb host innate immune responsesNeutrophils are resistant to Yersinia YopJ/P-induced apoptosis and are protected from ROS-mediated cell death by the type III secretion systemCaspase-8 and RIP kinases regulate bacteria-induced innate immune responses and cell death.Caspase-8 mediates caspase-1 processing and innate immune defense in response to bacterial blockade of NF-κB and MAPK signalingImmune evasion by Yersinia enterocolitica: differential targeting of dendritic cell subpopulations in vivo.Synthetic glycosidated phospholipids induce apoptosis through activation of FADD, caspase-8 and the mitochondrial death pathway.YopJ-induced caspase-1 activation in Yersinia-infected macrophages: independent of apoptosis, linked to necrosis, dispensable for innate host defense.Pathogen-mediated proteolysis of the cell death regulator RIPK1 and the host defense modulator RIPK2 in human aortic endothelial cells.Early apoptosis of macrophages modulated by injection of Yersinia pestis YopK promotes progression of primary pneumonic plaguePathogenesis of Y. enterocolitica and Y. pseudotuberculosis in Human Yersiniosis.Activity of Uncleaved Caspase-8 Controls Anti-bacterial Immune Defense and TLR-Induced Cytokine Production Independent of Cell DeathSubversion of inflammasome activation and pyroptosis by pathogenic bacteria.The diverse role of RIP kinases in necroptosis and inflammation.The influence of infectious factors on dendritic cell apoptosis.Activation and Evasion of Inflammasomes by Yersinia.Luteolin induces apoptosis by activating Fas signaling pathway at the receptor level in laryngeal squamous cell line Hep-2 cells.Cell-Extrinsic TNF Collaborates with TRIF Signaling To Promote Yersinia-Induced Apoptosis.RIPK1-dependent apoptosis bypasses pathogen blockade of innate signaling to promote immune defense.Severe, but not mild heat-shock treatment induces immunogenic cell death in cancer cells.Enhanced survival of BCG-stimulated dendritic cells: involvement of anti-apoptotic proteins and NF-κB.
P2860
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P2860
Catalytically active Yersinia outer protein P induces cleavage of RIP and caspase-8 at the level of the DISC independently of death receptors in dendritic cells.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Catalytically active Yersinia ...... receptors in dendritic cells.
@en
type
label
Catalytically active Yersinia ...... receptors in dendritic cells.
@en
prefLabel
Catalytically active Yersinia ...... receptors in dendritic cells.
@en
P2093
P2860
P1433
P1476
Catalytically active Yersinia ...... h receptors in dendritic cells
@en
P2093
Ingo B Autenrieth
Irena Adkins
Kathleen Richter
Klaus Ruckdeschel
Sabine Gröbner
Sebastian Schulz
Stefan Borgmann
P2860
P2888
P304
P356
10.1007/S10495-007-0100-X
P577
2007-10-01T00:00:00Z