Placental cytokines and the pathogenesis of preeclampsia.
about
Interventions for preventing pre-eclampsia and its consequences: generic protocolHypoxia up-regulated angiogenin and down-regulated vascular cell adhesion molecule-1 expression and secretion in human placental trophoblastsG-Protein-coupled receptors as potential drug candidates in preeclampsia: targeting the relaxin/insulin-like family peptide receptor 1 for treatment and preventionBiological Tools to Study the Effects of Environmental Contaminants at the Feto-Maternal InterfacePreeclampsia: long-term consequences for vascular healthCytokines as key players in the pathophysiology of preeclampsiaElucidating immune mechanisms causing hypertension during pregnancyThe renal circulation in normal pregnancy and preeclampsia: is there a place for relaxin?Innate immunity, decidual cells, and preeclampsiaVascular adaptation in pregnancy and endothelial dysfunction in preeclampsiaRelease of tumor necrosis factor-alpha and prostanoids in whole blood cultures after in vivo exposure to low-dose aspirinIron behaving badly: inappropriate iron chelation as a major contributor to the aetiology of vascular and other progressive inflammatory and degenerative diseasesPregnant rats treated with a high-fat/prooxidant Western diet with ANG II and TNF-α are resistant to elevations in blood pressure and renal oxidative stressAnalytical approaches to detect maternal/fetal genotype incompatibilities that increase risk of pre-eclampsia.Effects of hypoxia-inducible factor-1alpha overexpression in pregnant mice: possible implications for preeclampsia and intrauterine growth restrictionPlacenta growth factor: potential role in pregnancy.Down-regulation of TIMP3 leads to increase in TACE expression and TNFα production by placental trophoblast cells.Effects of 17-hydroxyprogesterone on tumor necrosis factor-alpha-induced hypertension during pregnancyResveratrol inhibits release of soluble fms-like tyrosine kinase (sFlt-1) and soluble endoglin and improves vascular dysfunction - implications as a preeclampsia treatment.The relationship of hypovitaminosis D and IL-6 in preeclampsiaProgesterone blunts vascular endothelial cell secretion of endothelin-1 in response to placental ischemiaAngiotensin receptor agonistic autoantibody-mediated tumor necrosis factor-alpha induction contributes to increased soluble endoglin production in preeclampsiaVascular and cellular calcium in normal and hypertensive pregnancyMaternal plasma concentrations of the soluble tumor necrosis factor receptor 2 are increased prior to the diagnosis of preeclampsia.The role of inflammation in the pathology of preeclampsiaHypoxia-inducible factor-1 mediates the biological effects of oxygen on human trophoblast differentiation through TGFbeta(3)Progesterone supplementation attenuates hypertension and the autoantibody to the angiotensin II type I receptor in response to elevated interleukin-6 during pregnancy.Cytokines and placental macrophages in regulation of birth activity.Plasma soluble endoglin concentration in pre-eclampsia is associated with an increased impedance to flow in the maternal and fetal circulations.Plasma from preeclamptic women increases blood-brain barrier permeability: role of vascular endothelial growth factor signaling.Regulation of sFlt-1 and VEGF secretion by adenosine under hypoxic conditions in rat placental villous explants.Placental tumor necrosis factor alpha but not gamma interferon is associated with placental malaria and low birth weight in Malawian women.Maternal plasma concentrations of sST2 and angiogenic/anti-angiogenic factors in preeclampsia.Maternal circulating TNF-alpha levels are highly correlated with IL-10 levels, but not IL-6 and IL-8 levels, in women with pre-eclampsia.The Role of Obesity in Preeclampsia.Association of Dll4/notch and HIF-1a -VEGF signaling in the angiogenesis of missed abortion.Upregulation of long noncoding RNA SPRY4-IT1 modulates proliferation, migration, apoptosis, and network formation in trophoblast cells HTR-8SV/neoExcessive stimulation of poly(ADP-ribosyl)ation contributes to endothelial dysfunction in pre-eclampsia.Secretion of tumor necrosis factor-alpha from human placental tissues induced by hypoxia-reoxygenation causes endothelial cell activation in vitro: a potential mediator of the inflammatory response in preeclampsia.Mesenchymal stem cells ameliorate Th1-induced pre-eclampsia-like symptoms in mice via the suppression of TNF-α expression.
P2860
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P2860
Placental cytokines and the pathogenesis of preeclampsia.
description
1997 nî lūn-bûn
@nan
1997年の論文
@ja
1997年論文
@yue
1997年論文
@zh-hant
1997年論文
@zh-hk
1997年論文
@zh-mo
1997年論文
@zh-tw
1997年论文
@wuu
1997年论文
@zh
1997年论文
@zh-cn
name
Placental cytokines and the pathogenesis of preeclampsia.
@en
type
label
Placental cytokines and the pathogenesis of preeclampsia.
@en
prefLabel
Placental cytokines and the pathogenesis of preeclampsia.
@en
P2860
P1476
Placental cytokines and the pathogenesis of preeclampsia.
@en
P2093
P2860
P304
P356
10.1111/J.1600-0897.1997.TB00222.X
P577
1997-03-01T00:00:00Z