about
Amylin at the interface between metabolic and neurodegenerative disordersOral Treatment with the Ghrelin Receptor Agonist HM01 Attenuates Cachexia in Mice Bearing Colon-26 (C26) TumorsCentral amylin acts as an adiposity signal to control body weight and energy expenditureRoux-en-Y gastric bypass surgery in rats alters gut microbiota profile along the intestine.Hypertrophy dependent doubling of L-cells in Roux-en-Y gastric bypass operated rats.The Sirt1 activator SRT3025 provides atheroprotection in Apoe-/- mice by reducing hepatic Pcsk9 secretion and enhancing Ldlr expression.The physiology underlying Roux-en-Y gastric bypass: a status report.Interleukin-6 contributes to early fasting-induced free fatty acid mobilization in mice.Physiological mechanisms behind Roux-en-Y gastric bypass surgery.Gastric bypass reduces fat intake and preference.Amylin-induced central IL-6 production enhances ventromedial hypothalamic leptin signaling.Translational value of animal models of obesity-Focus on dogs and cats.Roux-en-Y gastric bypass operation in rats.Overview of animal models of obesity.Longitudinal evaluation of serum pancreatic enzymes and ultrasonographic findings in diabetic cats without clinically relevant pancreatitis at diagnosis.Intensive intravenous infusion of insulin in diabetic cats.Acute peripheral GLP-1 receptor agonism or antagonism does not alter energy expenditure in rats after Roux-en-Y gastric bypassRoux-en-Y gastric bypass in rats increases sucrose taste-related motivated behavior independent of pharmacological GLP-1-receptor modulation.Brainstem mechanisms of amylin-induced anorexia.The role of amylin in the control of energy homeostasis.Amylinergic control of food intake in lean and obese rodents.Where to begin and where to end? Preoperative assessment for patients undergoing metabolic surgery.Reduced fasting-induced activation of hypothalamic arcuate neurons is associated with hyperleptinemia and increased leptin sensitivity in obese mice.Amylin: Pharmacology, Physiology, and Clinical Potential.Gastric bypass in rats does not decrease appetitive behavior towards sweet or fatty fluids despite blunting preferential intake of sugar and fatRenal morphology in cats with diabetes mellitus.Ghrelin-induced hypothermia: a physiological basis but no clinical risk.Predictors of clinical remission in cats with diabetes mellitus.Effect of hyperlipidemia on 11β-hydroxysteroid-dehydrogenase, glucocorticoid receptor, and leptin expression in insulin-sensitive tissues of cats.Feline pancreatic islet-like clusters and insulin producing cells express functional Toll-like receptors (TLRs).Basal plasma levels of insulin, leptin, ghrelin, and amylin do not signal adiposity in rats recovering from forced overweight.Noradrenergic neurons of the area postrema mediate amylin's hypophagic action.Involvement of nitric oxide in lipopolysaccharide induced anorexia.Toll-like receptor 2-deficient mice are protected from insulin resistance and beta cell dysfunction induced by a high-fat diet.Identification of central projections from amylin-activated neurons to the lateral hypothalamus.10-day hyperlipidemic clamp in cats: effects on insulin sensitivity, inflammation, and glucose metabolism-related genes.Effects of glucagon-like peptide 1 and oxyntomodulin on neuronal activity of ghrelin-sensitive neurons in the hypothalamic arcuate nucleus.Hyperglycaemia but not hyperlipidaemia decreases serum amylase and increases neutrophils in the exocrine pancreas of cats.Postprandial response of plasma insulin, amylin and acylated ghrelin to various test meals in lean and obese cats.Evaluation of a novel real-time continuous glucose-monitoring system for use in cats.
P50
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P50
description
hulumtues
@sq
researcher
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wetenschapper
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հետազոտող
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name
Thomas A Lutz
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Thomas A Lutz
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Thomas A Lutz
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Thomas A Lutz
@nl
Thomas A Lutz
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type
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Thomas A Lutz
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Thomas A Lutz
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Thomas A Lutz
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Thomas A Lutz
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Thomas A Lutz
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Thomas Lutz
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Thomas A Lutz
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Thomas A Lutz
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Thomas A Lutz
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Thomas A Lutz
@nl
Thomas A Lutz
@sl
P106
P21
P31
P496
0000-0002-5056-8548