Inflammation induced by innate immunity in the central nervous system leads to primary astrocyte dysfunction followed by demyelination.
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Connexin 43 astrocytopathy linked to rapidly progressive multiple sclerosis and neuromyelitis opticaMultiple sclerosis: molecular mechanisms and therapeutic opportunitiesSystemic inflammation disrupts oligodendrocyte gap junctions and induces ER stress in a model of CNS manifestations of X-linked Charcot-Marie-Tooth disease.Aquaporins in the Spinal CordPotassium channel KIR4.1 as an immune target in multiple sclerosisNeuromyelitis optica lesions may inform multiple sclerosis heterogeneity debateThe ubiquitin proteasome system in glia and its role in neurodegenerative diseasesToll-like receptors 2 and 3 agonists differentially affect oligodendrocyte survival, differentiation, and myelin membrane formationOptic neuritis in neuromyelitis optica.Mitochondrial dysfunction is an important cause of neurological deficits in an inflammatory model of multiple sclerosis.Regulation of gap junction channels by infectious agents and inflammation in the CNS.Neonatal astrocyte damage is sufficient to trigger progressive striatal degeneration in a rat model of glutaric acidemia-I.Presence of six different lesion types suggests diverse mechanisms of tissue injury in neuromyelitis optica11C-acetate PET imaging in patients with multiple sclerosis.Early disruption of glial communication via connexin gap junction in multiple sclerosis, Baló's disease and neuromyelitis optica.Early loss of oligodendrocytes in human and experimental neuromyelitis optica lesions.Neuromyelitis optica unique area postrema lesions: nausea, vomiting, and pathogenic implicationsComparison of clinical characteristics between neuromyelitis optica spectrum disorders with and without spinal cord atrophy.Oxidative damage in multiple sclerosis lesions.Specific densified regions in the postlaminar human glaucomatous optic nerve.Markedly increased IP-10 production by blood-brain barrier in neuromyelitis opticaNew insights into neuromyelitis opticaNADPH oxidase expression in active multiple sclerosis lesions in relation to oxidative tissue damage and mitochondrial injury.Subcutaneous Transplantation of Neural Precursor Cells in Experimental Autoimmune Encephalomyelitis Reduces Chemotactic Signals in the Central Nervous SystemBrain connexins in demyelinating diseases: therapeutic potential of glial targetsPossible involvement of TLRs and hemichannels in stress-induced CNS dysfunction via mastocytes, and glia activation.Cause and prevention of demyelination in a model multiple sclerosis lesion.Intrastriatal injection of interleukin-1 beta triggers the formation of neuromyelitis optica-like lesions in NMO-IgG seropositive ratsRegulation of astrocyte glutamine synthetase in epilepsyEvidence of aquaporin involvement in human central pontine myelinolysis.Multiple sclerosis: experimental models and reality.Reactive astrocytes as therapeutic targets for CNS disorders.Mechanisms of neurodegeneration shared between multiple sclerosis and Alzheimer's disease.Modulation of connexin signaling by bacterial pathogens and their toxins.Review: the architecture of inflammatory demyelinating lesions: implications for studies on pathogenesis.The molecular basis of neurodegeneration in multiple sclerosis.Oligodendrocytes and the early multiple sclerosis lesion.Connexins-mediated glia networking impacts myelination and remyelination in the central nervous system.Mechanisms of white matter damage in multiple sclerosis.The role of microglial activation in disease progression.
P2860
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P2860
Inflammation induced by innate immunity in the central nervous system leads to primary astrocyte dysfunction followed by demyelination.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
2010年论文
@zh
2010年论文
@zh-cn
name
Inflammation induced by innate ...... ion followed by demyelination.
@en
type
label
Inflammation induced by innate ...... ion followed by demyelination.
@en
prefLabel
Inflammation induced by innate ...... ion followed by demyelination.
@en
P2093
P2860
P50
P1476
Inflammation induced by innate ...... tion followed by demyelination
@en
P2093
Hans Lassmann
Kazuo Fujihara
Marie-Therese Fischer
Rakhi Sharma
Tatsuro Misu
P2860
P2888
P304
P356
10.1007/S00401-010-0704-Z
P577
2010-06-08T00:00:00Z