MAD2B contributes to podocyte injury of diabetic nephropathy via inducing cyclin B1 and Skp2 accumulation.
about
Cell cycle re-entry sensitizes podocytes to injury induced death.MDM2 mediates fibroblast activation and renal tubulointerstitial fibrosis via a p53-independent pathway.MAD2B promotes tubular epithelial-to-mesenchymal transition and renal tubulointerstitial fibrosis via Skp2.MDM2 is implicated in high-glucose-induced podocyte mitotic catastrophe via Notch1 signalling.Deletion of diacylglycerol-responsive TRPC genes attenuates diabetic nephropathy by inhibiting activation of the TGFβ1 signaling pathway.MAD2B-mediated SnoN downregulation is implicated in fibroblast activation and tubulointerstitial fibrosis.
P2860
MAD2B contributes to podocyte injury of diabetic nephropathy via inducing cyclin B1 and Skp2 accumulation.
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
@zh
2015年论文
@zh-cn
name
MAD2B contributes to podocyte ...... clin B1 and Skp2 accumulation.
@en
type
label
MAD2B contributes to podocyte ...... clin B1 and Skp2 accumulation.
@en
prefLabel
MAD2B contributes to podocyte ...... clin B1 and Skp2 accumulation.
@en
P2093
P2860
P1476
MAD2B contributes to podocyte ...... clin B1 and Skp2 accumulation.
@en
P2093
Chun Zhang
Fang-Fang He
Xian-Fang Meng
Xiu-Juan Tian
P2860
P304
P356
10.1152/AJPRENAL.00409.2014
P50
P577
2015-01-28T00:00:00Z