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Caspase-activated DNase is required for maintenance of tolerance to lupus nuclear autoantigensThe Bruton tyrosine kinase inhibitor PCI-32765 blocks B-cell activation and is efficacious in models of autoimmune disease and B-cell malignancyNuclear Factor Erythroid 2-related Factor 2 Deficiency Exacerbates Lupus Nephritis in B6/lpr mice by Regulating Th17 Cell FunctionPrevention of murine antiphospholipid syndrome by BAFF blockadeB cell depletion with anti-CD79 mAbs ameliorates autoimmune disease in MRL/lpr mice.Similarities and differences between selective and nonselective BAFF blockade in murine SLEHDAC inhibition in lupus models.Amino acid metabolism inhibits antibody-driven kidney injury by inducing autophagy.Identification of stage-specific genes associated with lupus nephritis and response to remission induction in (NZB × NZW)F1 and NZM2410 mice.Immune-Mediated Nephropathy and Systemic Autoimmunity in Mice Does Not Require Receptor Interacting Protein Kinase 3 (RIPK3).Pathology and protection in nephrotoxic nephritis is determined by selective engagement of specific Fc receptors.A novel mouse with B cells but lacking serum antibody reveals an antibody-independent role for B cells in murine lupus.Interleukin-17 cytokines are critical in development of fatal lupus glomerulonephritisActivated renal macrophages are markers of disease onset and disease remission in lupus nephritis.Selective Memory to Apoptotic Cell-Derived Self-Antigens with Implications for Systemic Lupus Erythematosus DevelopmentPotential impact of B cells on T cell function in multiple sclerosisTCF1 deficiency ameliorates autoimmune lymphoproliferative syndrome (ALPS)-like phenotypes of lpr/lpr mice.Pre-treatment with Lactobacillus plantarum prevents severe pathogenesis in mice infected with Leptospira interrogans and may be associated with recruitment of myeloid cells.Increased B cell deletion and significantly reduced auto-antibody titre due to premature expression of human complement receptor 2 (CR2, CD21).Anti-tumor necrosis factor α treatment of interferon-α-induced murine lupus nephritis reduces the renal macrophage response but does not alter glomerular immune complex formation.Selective blockade of BAFF for the prevention and treatment of systemic lupus erythematosus nephritis in NZM2410 mice.Stabilized β-Catenin Ameliorates ALPS-Like Symptoms of B6/lpr Mice.Estrogen Receptor α Signaling Exacerbates Immune-Mediated Nephropathies through Alteration of Metabolic Activity.iRhom2 promotes lupus nephritis through TNF-α and EGFR signaling.The origin of anti-nuclear antibodies in bcl-2 transgenic mice.Alterations in splenic architecture and the localization of anti-double-stranded DNA B cells in aged mice.
P2860
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P2860
description
1997 nî lūn-bûn
@nan
1997年の論文
@ja
1997年論文
@yue
1997年論文
@zh-hant
1997年論文
@zh-hk
1997年論文
@zh-mo
1997年論文
@zh-tw
1997年论文
@wuu
1997年论文
@zh
1997年论文
@zh-cn
name
The roles of B cells in MRL/lpr murine lupus.
@en
type
label
The roles of B cells in MRL/lpr murine lupus.
@en
prefLabel
The roles of B cells in MRL/lpr murine lupus.
@en
P2093
P2860
P1476
The roles of B cells in MRL/lpr murine lupus.
@en
P2093
P2860
P356
10.1111/J.1749-6632.1997.TB52046.X
P407
P577
1997-04-01T00:00:00Z