Craniofacial skeletal defects of adult zebrafish Glypican 4 (knypek) mutants. - Wikidata - awgldk - TriplyDB

Craniofacial skeletal defects of adult zebrafish Glypican 4 (knypek) mutants.

Craniofacial skeletal defects of adult zebrafish Glypican 4 (knypek) mutants.

http://www.wikidata.org/entity/Q41520660

about

Bentho-pelagic divergence of cichlid feeding architecture was prodigious and consistent during multiple adaptive radiations within African rift-lakes Dchs1-Fat4 regulation of polarized cell behaviours during skeletal morphogenesis.On the roles and regulation of chondroitin sulfate and heparan sulfate in zebrafish pharyngeal cartilage morphogenesis Identification of an evolutionarily conserved regulatory element of the zebrafish col2a1a gene.Exploring the effects of gene dosage on mandible shape in mice as a model for studying the genetic basis of natural variation Prediction of regulatory networks in mouse abdominal wall Heparan sulfate proteoglycans.Proteomics analysis of the zebrafish skeletal extracellular matrix.Planar cell polarity signaling in craniofacial development.A role of glypican4 and wnt5b in chondrocyte stacking underlying craniofacial cartilage morphogenesis Developmental basis of phenotypic integration in two Lake Malawi cichlids Roles of Wnt pathway genes wls, wnt9a, wnt5b, frzb and gpc4 in regulating convergent-extension during zebrafish palate morphogenesis.Identification and expression analysis of zebrafish glypicans during embryonic development.Distinct requirements of wls, wnt9a, wnt5b and gpc4 in regulating chondrocyte maturation and timing of endochondral ossification.Syndecans in cartilage breakdown and synovial inflammation.Boning up on glypicans--opportunities for new insights into bone biology.Chondroitin sulphate and heparan sulphate sulphation motifs and their proteoglycans are involved in articular cartilage formation during human foetal knee joint development.Kinesin-1 promotes chondrocyte maintenance during skeletal morphogenesis.Tetrahydroxystilbene glucoside isolated from Polygonum multiflorum Thunb. demonstrates osteoblast differentiation promoting activity.Wnt5b regulates mesenchymal cell aggregation and chondrocyte differentiation through the planar cell polarity pathway.Cartilage ultrastructure in proteoglycan-deficient zebrafish mutants brings to light new candidate genes for human skeletal disorders.Modulating Glypican4 suppresses tumorigenicity of embryonic stem cells while preserving self-renewal and pluripotency.

P2860

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P2860

Craniofacial skeletal defects of adult zebrafish Glypican 4 (knypek) mutants.

http://www.wikidata.org/entity/Q41520660

description

2009 nî lūn-bûn

@nan

2009年の論文

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2009年論文

@yue

2009年論文

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2009年論文

@zh-hk

2009年論文

@zh-mo

2009年論文

@zh-tw

2009年论文

@wuu

2009年论文

@zh

2009年论文

@zh-cn

name

Craniofacial skeletal defects of adult zebrafish Glypican 4 (knypek) mutants.

@en

type

label

Craniofacial skeletal defects of adult zebrafish Glypican 4 (knypek) mutants.

@en

prefLabel

Craniofacial skeletal defects of adult zebrafish Glypican 4 (knypek) mutants.

@en

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Developmental Dynamics

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Craniofacial skeletal defects of adult zebrafish Glypican 4 (knypek) mutants

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P2093

Angela Huang

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Elizabeth E LeClair

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Jolanta M Topczewska

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Stephanie R Mui

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P2860

A zebrafish screen for craniofacial mutants identifies wdr68 as a highly conserved gene required for endothelin-1 expression

Glypican LON-2 is a conserved negative regulator of BMP-like signaling in Caenorhabditis elegans

Mutations in GPC3, a glypican gene, cause the Simpson-Golabi-Behmel overgrowth syndrome

Glypican-3-deficient mice exhibit developmental overgrowth and some of the abnormalities typical of Simpson-Golabi-Behmel syndrome

Mutagenesis strategies in zebrafish for identifying genes involved in development and disease

A Wnt5a pathway underlies outgrowth of multiple structures in the vertebrate embryo

glypican-3 controls cellular responses to Bmp4 in limb patterning and skeletal development

Overgrowth of a mouse model of the Simpson-Golabi-Behmel syndrome is independent of IGF signaling

Altered hematopoiesis in glypican-3-deficient mice results in decreased osteoclast differentiation and a delay in endochondral ossification

Glypican-3 inhibits Hedgehog signaling during development by competing with patched for Hedgehog binding

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2550-2563

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10.1002/DVDY.22086

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10

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238

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Jacek Topczewski

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26834602

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19777561

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3011836

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