Peptide SS-31 upregulates frataxin expression and improves the quality of mitochondria: implications in the treatment of Friedreich ataxia.

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SS-31 Provides Neuroprotection by Reversing Mitochondrial Dysfunction after Traumatic Brain Injury

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Peptide SS-31 upregulates frataxin expression and improves the quality of mitochondria: implications in the treatment of Friedreich ataxia.

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http://www.wikidata.org/entity/Q41548664

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年學術文章

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2017年學術文章

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2017年學術文章

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name

Peptide SS-31 upregulates frat ...... reatment of Friedreich ataxia.

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Peptide SS-31 upregulates frat ...... reatment of Friedreich ataxia.

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Peptide SS-31 upregulates frat ...... reatment of Friedreich ataxia.

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P1476

Peptide SS-31 upregulates frat ...... reatment of Friedreich ataxia.

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P2093

Hongting Zhao

http://www.w3.org/2001/XMLSchema#string

Huihui Li

http://www.w3.org/2001/XMLSchema#string

Jing Wu

http://www.w3.org/2001/XMLSchema#string

Jiping Chen

http://www.w3.org/2001/XMLSchema#string

Kuanyu Li

http://www.w3.org/2001/XMLSchema#string

Meng Zhang

http://www.w3.org/2001/XMLSchema#string

Shuangying Hao

http://www.w3.org/2001/XMLSchema#string

Tong Qiao

http://www.w3.org/2001/XMLSchema#string

P2860

Iron-sulfur cluster biosynthesis. Characterization of frataxin as an iron donor for assembly of [2Fe-2S] clusters in ISU-type proteins

Iron-dependent regulation of frataxin expression: implications for treatment of Friedreich ataxia

Human frataxin is an allosteric switch that activates the Fe-S cluster biosynthetic complex

Aconitase and mitochondrial iron-sulphur protein deficiency in Friedreich ataxia

Activating frataxin expression by repeat-targeted nucleic acids

Friedreich's ataxia: autosomal recessive disease caused by an intronic GAA triplet repeat expansion

Targeting mitochondrial cardiolipin and the cytochrome c/cardiolipin complex to promote electron transport and optimize mitochondrial ATP synthesis

First-in-class cardiolipin-protective compound as a therapeutic agent to restore mitochondrial bioenergetics

The mitochondrial-targeted compound SS-31 re-energizes ischemic mitochondria by interacting with cardiolipin

Friedreich's ataxia, no changes in mitochondrial labile iron in human lymphoblasts and fibroblasts: a decrease in antioxidative capacity?

P2888

s41598-017-10320-2

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9840

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scholarly article

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10.1038/S41598-017-10320-2

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English

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http://www.w3.org/2001/XMLSchema#string

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Chuanhui Song

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2017-08-29T00:00:00Z

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1091331798

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28852135

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mitochondrion

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5575096

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Peptide SS-31 upregulates frataxin expression and improves the quality of mitochondria: implications in the treatment of Friedreich ataxia.

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P2860

P2860

Peptide SS-31 upregulates frataxin expression and improves the quality of mitochondria: implications in the treatment of Friedreich ataxia.

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