Excitotoxic mechanisms of neurodegeneration in transmissible spongiform encephalopathies.
about
Regional susceptibility to domoic acid in primary astrocyte cells cultured from the brain stem and hippocampus.Overexpression of wild-type murine tau results in progressive tauopathy and neurodegeneration.From toxins targeting ligand gated ion channels to therapeutic molecules.Statins are ineffective at reducing neuroinflammation or prolonging survival in scrapie-infected mice.Increased c-Fos protein in the brains of scrapie-infected SAMP8, SAMR1, AKR and C57BL mice.Expression of excitatory amino acid transporter-1 (EAAT-1) in brain macrophages and microglia of patients with prion diseases.Excitotoxicity through NMDA receptors mediates cerebellar granule neuron apoptosis induced by prion protein 90-231 fragment.
P2860
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P2860
Excitotoxic mechanisms of neurodegeneration in transmissible spongiform encephalopathies.
description
1997 nî lūn-bûn
@nan
1997年の論文
@ja
1997年論文
@yue
1997年論文
@zh-hant
1997年論文
@zh-hk
1997年論文
@zh-mo
1997年論文
@zh-tw
1997年论文
@wuu
1997年论文
@zh
1997年论文
@zh-cn
name
Excitotoxic mechanisms of neurodegeneration in transmissible spongiform encephalopathies.
@en
type
label
Excitotoxic mechanisms of neurodegeneration in transmissible spongiform encephalopathies.
@en
prefLabel
Excitotoxic mechanisms of neurodegeneration in transmissible spongiform encephalopathies.
@en
P2860
P1476
Excitotoxic mechanisms of neurodegeneration in transmissible spongiform encephalopathies.
@en
P2860
P304
P356
10.1111/J.1749-6632.1997.TB48429.X
P407
P577
1997-10-01T00:00:00Z