Occupancy of CD11b/CD18 (Mac-1) divalent ion binding site(s) induces leukocyte adhesion.
about
Pericytes regulate vascular basement membrane remodeling and govern neutrophil extravasation during inflammationGlycosyltransferase-programmed stereosubstitution (GPS) to create HCELL: engineering a roadmap for cell migrationBordetella adenylate cyclase toxin is a unique ligand of the integrin complement receptor 3The non-receptor tyrosine kinase Lyn controls neutrophil adhesion by recruiting the CrkL-C3G complex and activating Rap1 at the leading edgeSmall molecule agonists of integrin CD11b/CD18 do not induce global conformational changes and are significantly better than activating antibodies in reducing vascular injury.Small molecule-mediated activation of the integrin CD11b/CD18 reduces inflammatory disease.Micromechanical tests of adhesion dynamics between neutrophils and immobilized ICAM-1Neutrophil adhesive contact dependence on impingement force.Shifts in the concentrations of magnesium and calcium in early porcine and rat wound fluids activate the cell migratory responseRegulation of cellular interactions with laminin by integrin cytoplasmic domains: the A and B structural variants of the alpha 6 beta 1 integrin differentially modulate the adhesive strength, morphology, and migration of macrophages.A subpopulation of Mac-1 (CD11b/CD18) molecules mediates neutrophil adhesion to ICAM-1 and fibrinogenIntegrin cytoplasmic domains mediate inside-out signal transduction.Two functional states of the CD11b A-domain: correlations with key features of two Mn2+-complexed crystal structures.Differential roles for alpha(M)beta(2) integrin clustering or activation in the control of apoptosis via regulation of akt and ERK survival mechanismsUrokinase receptor (CD87) regulates leukocyte recruitment via beta 2 integrins in vivo.Activation of human neutrophil Mac-1 by anion substitutionThe urokinase receptor (CD87) facilitates CD11b/CD18-mediated adhesion of human monocytesAgonist leukadherin-1 increases CD11b/CD18-dependent adhesion via membrane tethers.Cytoplasmic tails of beta 1, beta 2, and beta 7 integrins differentially regulate LFA-1 function in K562 cells.Extracellular Ca2+ modulates leukocyte function-associated antigen-1 cell surface distribution on T lymphocytes and consequently affects cell adhesion.Chemoattractants induce a rapid and transient upregulation of monocyte alpha4 integrin affinity for vascular cell adhesion molecule 1 which mediates arrest: an early step in the process of emigration.
P2860
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P2860
Occupancy of CD11b/CD18 (Mac-1) divalent ion binding site(s) induces leukocyte adhesion.
description
1991 nî lūn-bûn
@nan
1991年の論文
@ja
1991年論文
@yue
1991年論文
@zh-hant
1991年論文
@zh-hk
1991年論文
@zh-mo
1991年論文
@zh-tw
1991年论文
@wuu
1991年论文
@zh
1991年论文
@zh-cn
name
Occupancy of CD11b/CD18 (Mac-1) divalent ion binding site(s) induces leukocyte adhesion.
@en
type
label
Occupancy of CD11b/CD18 (Mac-1) divalent ion binding site(s) induces leukocyte adhesion.
@en
prefLabel
Occupancy of CD11b/CD18 (Mac-1) divalent ion binding site(s) induces leukocyte adhesion.
@en
P1476
Occupancy of CD11b/CD18 (Mac-1) divalent ion binding site(s) induces leukocyte adhesion.
@en
P2093
Altieri DC
P304
P407
P577
1991-09-01T00:00:00Z