Various hypertrophic stimuli induce distinct phenotypes in cardiomyocytes.
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Focal adhesion kinase and p130Cas mediate both sarcomeric organization and activation of genes associated with cardiac myocyte hypertrophyMaternal nutrient restriction and the fetal left ventricle: decreased angiotensin receptor expressionDynamic Myofibrillar Remodeling in Live Cardiomyocytes under Static Stretch.Telokin expression is restricted to smooth muscle tissues during mouse developmentCardiac troponin T mutations result in allele-specific phenotypes in a mouse model for hypertrophic cardiomyopathyNetwork reconstruction and systems analysis of cardiac myocyte hypertrophy signaling.Multifaceted roles of miR-1s in repressing the fetal gene program in the heartCardiac hypertrophy: signal transduction, transcriptional adaptation, and altered growth control.Cardiac-specific overexpression of RhoA results in sinus and atrioventricular nodal dysfunction and contractile failure.Selective decrease of components of the creatine kinase system and ATP synthase complex in chronic Chagas disease cardiomyopathyAldosterone, a new appreciation of its role in heart failure.Down-regulation of replication factor C-40 (RFC40) causes chromosomal missegregation in neonatal and hypertrophic adult rat cardiac myocytes.Thyroid Hormone-Regulated Cardiac microRNAs are Predicted to Suppress Pathological Hypertrophic Signaling.Docosahexaenoic acid supplementation does not improve Western diet-induced cardiomyopathy in rats.The role of dietary fatty acids in predicting myocardial structure in fat-fed ratsCardiac gene expression profiling provides evidence for cytokinopathy as a molecular mechanism in Chagas' disease cardiomyopathy.Vascular failure: a hypothesis.Role for interleukin-1 beta in Trypanosoma cruzi-induced cardiomyocyte hypertrophy.Progenitor cell therapy in a porcine acute myocardial infarction model induces cardiac hypertrophy, mediated by paracrine secretion of cardiotrophic factors including TGFbeta1.Thyroid hormone drives fetal cardiomyocyte maturation.Expression profiling reveals distinct sets of genes altered during induction and regression of cardiac hypertrophyPathological situations characterized by altered actin isoform expression.Placement of an elastic biodegradable cardiac patch on a subacute infarcted heart leads to cellularization with early developmental cardiomyocyte characteristicsDilated cardiomyopathy and impaired cardiac hypertrophic response to angiotensin II in mice lacking FGF-2.Regression of copper-deficient heart hypertrophy: reduction in the size of hypertrophic cardiomyocytes.Molecular defects in cardiac myofibrillar proteins due to thyroid hormone imbalance and diabetes.Electrical and mechanical stimulation of cardiac cells and tissue constructs.Myofibrillar remodeling in cardiac hypertrophy, heart failure and cardiomyopathies.Stem cells and their derivatives: a renaissance in cardiovascular translational research.Strategies for the chemical and biological functionalization of scaffolds for cardiac tissue engineering: a review.Human-induced pluripotent stem cell-derived cardiomyocytes exhibit temporal changes in phenotype.microRNA-133a regulates cardiomyocyte proliferation and suppresses smooth muscle gene expression in the heart.Synergistic roles of neuregulin-1 and insulin-like growth factor-I in activation of the phosphatidylinositol 3-kinase pathway and cardiac chamber morphogenesis.Regulation of thyroid cell volumes and fluid transport: opposite effects of TSH and iodide on cultured cells.The protective role of peroxisome proliferator-activated receptor γ coactivator-1α in hyperthyroid cardiac hypertrophy.Toll-like receptor 2 regulates interleukin-1beta-dependent cardiomyocyte hypertrophy triggered by Trypanosoma cruzi.Demonstration of direct effects of growth hormone on neonatal cardiomyocytes.Regulation of Cyr61 gene expression by mechanical stretch through multiple signaling pathways.Load-dependent induction of apoptosis in multicellular myocardial preparations.Effects of gestational age on myocardial blood flow and coronary flow reserve in pressure-loaded ovine fetal hearts.
P2860
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P2860
Various hypertrophic stimuli induce distinct phenotypes in cardiomyocytes.
description
1997 nî lūn-bûn
@nan
1997年の論文
@ja
1997年論文
@yue
1997年論文
@zh-hant
1997年論文
@zh-hk
1997年論文
@zh-mo
1997年論文
@zh-tw
1997年论文
@wuu
1997年论文
@zh
1997年论文
@zh-cn
name
Various hypertrophic stimuli induce distinct phenotypes in cardiomyocytes.
@en
type
label
Various hypertrophic stimuli induce distinct phenotypes in cardiomyocytes.
@en
prefLabel
Various hypertrophic stimuli induce distinct phenotypes in cardiomyocytes.
@en
P2093
P356
P1476
Various hypertrophic stimuli induce distinct phenotypes in cardiomyocytes.
@en
P2093
Eppenberger HM
P2888
P304
P356
10.1007/S001090050182
P577
1997-11-01T00:00:00Z