Effect of IRAK-M on Airway Inflammation Induced by Cigarette Smoking.

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Effect of IRAK-M on Airway Inflammation Induced by Cigarette Smoking.

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2017 nî lūn-bûn

@nan

2017年の論文

@ja

2017年論文

@yue

2017年論文

@zh-hant

2017年論文

@zh-hk

2017年論文

@zh-mo

2017年論文

@zh-tw

2017年论文

@wuu

2017年论文

@zh

2017年论文

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Effect of IRAK-M on Airway Inflammation Induced by Cigarette Smoking.

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Item

label

Effect of IRAK-M on Airway Inflammation Induced by Cigarette Smoking.

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prefLabel

Effect of IRAK-M on Airway Inflammation Induced by Cigarette Smoking.

@en

P1476

Effect of IRAK-M on Airway Inflammation Induced by Cigarette Smoking.

@en

P2093

Haihong Gong

http://www.w3.org/2001/XMLSchema#string

Tao Liu

http://www.w3.org/2001/XMLSchema#string

Wei Chen

http://www.w3.org/2001/XMLSchema#string

Weixun Zhou

http://www.w3.org/2001/XMLSchema#string

P2860

IRAK-M is a negative regulator of Toll-like receptor signaling

Pathological versus protective functions of IL-22 in airway inflammation are regulated by IL-17A

IRAK-M is involved in the pathogenesis of early-onset persistent asthma

IRAK-M expression limits dendritic cell activation and proinflammatory cytokine production in response to Helicobacter pylori

The orphan nuclear receptor RORgammat directs the differentiation program of proinflammatory IL-17+ T helper cells

Origin and functions of pro-inflammatory cytokine producing Foxp3+ regulatory T cells

Interleukin-17 and type 17 helper T cells

Differential role for CD80 and CD86 in the regulation of the innate immune response in murine polymicrobial sepsis

Lung dendritic cell expression of maturation molecules increases with worsening chronic obstructive pulmonary disease.

Cigarette smoke promotes dendritic cell accumulation in COPD; a Lung Tissue Research Consortium study

P304

6506953

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P31

scholarly article

P356

10.1155/2017/6506953

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P478

2017

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P50

Jinming Gao

P577

2017-08-29T00:00:00Z

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P698

28951634

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P932

5603328

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