Comparative transcriptome analysis of muscular dystrophy models Large(myd), Dmd(mdx)/Large(myd) and Dmd(mdx): what makes them different?

about

Comparative transcriptome analysis of muscular dystrophy models Large(myd), Dmd(mdx)/Large(myd) and Dmd(mdx): what makes them different?

Item

http://www.wikidata.org/entity/Q41691404

description

2016 nî lūn-bûn

@nan

2016年の論文

@ja

2016年論文

@yue

2016年論文

@zh-hant

2016年論文

@zh-hk

2016年論文

@zh-mo

2016年論文

@zh-tw

2016年论文

@wuu

2016年论文

@zh

2016年论文

@zh-cn

name

Comparative transcriptome anal ...... x): what makes them different?

@en

type

Item

label

Comparative transcriptome anal ...... x): what makes them different?

@en

prefLabel

Comparative transcriptome anal ...... x): what makes them different?

@en

P1476

Comparative transcriptome anal ...... x): what makes them different?

@en

P2093

Camila F Almeida

http://www.w3.org/2001/XMLSchema#string

Mariz Vainzof

http://www.w3.org/2001/XMLSchema#string

Poliana Cm Martins

http://www.w3.org/2001/XMLSchema#string

P2860

Expression and DNA sequence analysis of a human embryonic skeletal muscle myosin heavy chain gene

The MyoD-inducible p204 protein overcomes the inhibition of myoblast differentiation by Id proteins.

Significance analysis of microarrays applied to the ionizing radiation response

Muscle regeneration in dystrophin-deficient mdx mice studied by gene expression profiling

Mutations in the human LARGE gene cause MDC1D, a novel form of congenital muscular dystrophy with severe mental retardation and abnormal glycosylation of alpha-dystroglycan

A chronic inflammatory response dominates the skeletal muscle molecular signature in dystrophin-deficient mdx mice

Matrix metalloproteinases: fold and function of their catalytic domains

Macrophage-specific metalloelastase (MMP-12) truncates and inactivates ELR+ CXC chemokines and generates CCL2, -7, -8, and -13 antagonists: potential role of the macrophage in terminating polymorphonuclear leukocyte influx

A skeletal muscle-specific enhancer regulated by factors binding to E and CArG boxes is present in the promoter of the mouse myosin light-chain 1A gene

Calpain-6 deficiency promotes skeletal muscle development and regeneration

P2888

ejhg.2016.16

P304

1301-1309

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1038/EJHG.2016.16

http://www.w3.org/2001/XMLSchema#string

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P577

2016-03-02T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P6179

1038215990

http://www.w3.org/2001/XMLSchema#string

P698

26932192

http://www.w3.org/2001/XMLSchema#string

P932

4989205

http://www.w3.org/2001/XMLSchema#string