Ampk phosphorylation of Ulk1 is required for targeting of mitochondria to lysosomes in exercise-induced mitophagy. - Wikidata - awgldk - TriplyDB

Ampk phosphorylation of Ulk1 is required for targeting of mitochondria to lysosomes in exercise-induced mitophagy.

Ampk phosphorylation of Ulk1 is required for targeting of mitochondria to lysosomes in exercise-induced mitophagy.

http://www.wikidata.org/entity/Q41701419

about

Possible roles for ATP release from RBCs exclude the cAMP-mediated Panx1 pathway.Mitophagy in maintaining skeletal muscle mitochondrial proteostasis and metabolic health with ageing.AMPK: guardian of metabolism and mitochondrial homeostasis.Exercise and Mitochondrial Dynamics: Keeping in Shape with ROS and AMPK.AMPK in skeletal muscle function and metabolism.Linking Cancer Cachexia-Induced Anabolic Resistance to Skeletal Muscle Oxidative Metabolism.The Role of gp130 in Basal and Exercise Trained Skeletal Muscle Mitochondrial Quality Control.Mitochondrial protein S-nitrosation protects against ischemia reperfusion-induced denervation at neuromuscular junction in skeletal muscle.Use the Protonmotive Force: Mitochondrial Uncoupling and Reactive Oxygen Species.Targeting AMPK Signaling as a Neuroprotective Strategy in Parkinson's Disease.Unclogging the garbage disposal: how exercise may improve mitochondria in ageing skeletal muscle

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P2860

Ampk phosphorylation of Ulk1 is required for targeting of mitochondria to lysosomes in exercise-induced mitophagy.

http://www.wikidata.org/entity/Q41701419

description

2017 nî lūn-bûn

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2017年の論文

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2017年論文

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2017年論文

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2017年論文

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2017年论文

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2017年论文

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2017年论文

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name

Ampk phosphorylation of Ulk1 i ...... in exercise-induced mitophagy.

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Ampk phosphorylation of Ulk1 i ...... in exercise-induced mitophagy.

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Ampk phosphorylation of Ulk1 i ...... in exercise-induced mitophagy.

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S41467-017-00520-9

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Nature Communications

P1476

Ampk phosphorylation of Ulk1 i ...... in exercise-induced mitophagy.

@en

P2093

Bevan M Lewellen

http://www.w3.org/2001/XMLSchema#string

Carleigh C Fisher

http://www.w3.org/2001/XMLSchema#string

Jeffrey J Saucerman

http://www.w3.org/2001/XMLSchema#string

Joshua C Drake

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Karen A Ryall

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Laurie J Goodyear

http://www.w3.org/2001/XMLSchema#string

Mei Zhang

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Rebecca J Wilson

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Rhianna C Laker

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Vitor A Lira

http://www.w3.org/2001/XMLSchema#string

P2860

Nix is a selective autophagy receptor for mitochondrial clearance

PINK1/Parkin-mediated mitophagy is dependent on VDAC1 and p62/SQSTM1

Dephosphorylation by calcineurin regulates translocation of Drp1 to mitochondria

Endoplasmic reticulum stress: cell life and death decisions.

Exercise-induced BCL2-regulated autophagy is required for muscle glucose homeostasis

Physical exercise stimulates autophagy in normal skeletal muscles but is detrimental for collagen VI-deficient muscles

How mitochondria produce reactive oxygen species

Mitochondrial Quality Control and Muscle Mass Maintenance

Autophagy is essential to support skeletal muscle plasticity in response to endurance exercise

Metabolism. AMP-activated protein kinase mediates mitochondrial fission in response to energy stress.

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28916822

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phosphorylation

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5601463

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