Implication of the glutamate-cystine antiporter xCT in schizophrenia cases linked to impaired GSH synthesis.

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Implication of the glutamate-cystine antiporter xCT in schizophrenia cases linked to impaired GSH synthesis.

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http://www.wikidata.org/entity/Q41701597

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2017 nî lūn-bûn

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2017年の論文

@ja

2017年論文

@yue

2017年論文

@zh-hant

2017年論文

@zh-hk

2017年論文

@zh-mo

2017年論文

@zh-tw

2017年论文

@wuu

2017年论文

@zh

2017年论文

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Implication of the glutamate-c ...... ked to impaired GSH synthesis.

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label

Implication of the glutamate-c ...... ked to impaired GSH synthesis.

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prefLabel

Implication of the glutamate-c ...... ked to impaired GSH synthesis.

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P1476

Implication of the glutamate-c ...... ked to impaired GSH synthesis.

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P2093

A Monin

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C Ferrari

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K Do

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M Fournier

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P Conus

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P S Baumann

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P2860

CD44 variant regulates redox status in cancer cells by stabilizing the xCT subunit of system xc(-) and thereby promotes tumor growth

Keap1 is a redox-regulated substrate adaptor protein for a Cul3-dependent ubiquitin ligase complex.

Oxidative stress sensor Keap1 functions as an adaptor for Cul3-based E3 ligase to regulate proteasomal degradation of Nrf2

Keap1 represses nuclear activation of antioxidant responsive elements by Nrf2 through binding to the amino-terminal Neh2 domain

Thinking outside the cleft to understand synaptic activity: contribution of the cystine-glutamate antiporter (System xc-) to normal and pathological glutamatergic signaling

Exploration, normalization, and summaries of high density oligonucleotide array probe level data

An Nrf2/small Maf heterodimer mediates the induction of phase II detoxifying enzyme genes through antioxidant response elements

affy--analysis of Affymetrix GeneChip data at the probe level

The Keap1-BTB protein is an adaptor that bridges Nrf2 to a Cul3-based E3 ligase: oxidative stress sensing by a Cul3-Keap1 ligase

Assessment of glutamate transporter GLAST (EAAT1)-deficient mice for phenotypes relevant to the negative and executive/cognitive symptoms of schizophrenia.

P2888

s41537-017-0035-3

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scholarly article

P356

10.1038/S41537-017-0035-3

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P433

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P478

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P577

2017-09-18T00:00:00Z

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P6179

1091675692

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P698

28924227

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P921

schizophrenia

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5603608

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