Ovarian epithelial tumor growth promotion by follicle-stimulating hormone and inhibition of the effect by luteinizing hormone.
about
Mitogen-activated protein kinases in normal and (pre)neoplastic ovarian surface epitheliumControlled ovarian hyperstimulation for IVF: impact on ovarian, endometrial and cervical cancer--a systematic review and meta-analysisSex-steroid hormones, gonadotropin and ovarian carcinogenesis: a review of epidemiological and experimental data.Prohibitin: targeting peptide coupled to ovarian cancer, luteinization and TGF-β pathways.The Asn680Ser polymorphism of the follicle stimulating hormone receptor gene and ovarian cancer risk: a meta-analysis.Inhibitory role of prohibitin in human ovarian epithelial cancer.Comparison of expression profiles in ovarian epithelium in vivo and ovarian cancer identifies novel candidate genes involved in disease pathogenesis.Epithelial-to-Mesenchymal Transition in the Female Reproductive Tract: From Normal Functioning to Disease Pathology.Human chorionic gonadotropin and its relation to grade, stage and patient survival in ovarian cancer.The luteinizing hormone receptor: insights into structure-function relationships and hormone-receptor-mediated changes in gene expression in ovarian cancer cells.Glycodelin A is a prognostic marker to predict poor outcome in advanced stage ovarian cancer patients.The G-protein coupled estrogen receptor (GPER/GPR30) is a gonadotropin receptor dependent positive prognosticator in ovarian carcinoma patients.Effect of luteinizing hormone-induced prohibitin and matrix metalloproteinases on ovarian epithelial tumor cell proliferation.Microenvironment and pathogenesis of epithelial ovarian cancerFSH stimulates ovarian cancer cell growth by action on growth factor variant receptorDesmocollin 3 mediates follicle stimulating hormone-induced ovarian epithelial cancer cell proliferation by activating the EGFR/Akt signaling pathway.Follicle-Stimulating Hormone Receptor as a Target in the Redirected T-cell Therapy for Cancer.The insulin and igf-I pathway in endocrine glands carcinogenesisLuteinizing hormone inhibits cisplatin-induced apoptosis in human epithelial ovarian cancer cellsRole of endocrine and growth factors on the ovarian surface epithelium.A clinically applicable molecular classification for high-grade serous ovarian cancer based on hormone receptor expression.Targeting signaling pathways in epithelial ovarian cancer.Prediagnostic circulating follicle stimulating hormone concentrations and ovarian cancer risk.Ascites analysis by a microfluidic chip allows tumor-cell profiling.BRCA1 expression, proliferative and apoptotic activities in ovarian epithelial inclusions.The Role of Endocrine G Protein-Coupled Receptors in Ovarian Cancer Progression.Differential action of glycoprotein hormones: significance in cancer progression.Development of a mouse model of menopausal ovarian cancer.VEGF expression and enhanced production by gonadotropins in ovarian epithelial tumors.Anomalous endocrine feedback of peri-menopause in the etiology of type II ovarian cancer.Activation of the PI3K/AKT pathway mediates FSH-stimulated VEGF expression in ovarian serous cystadenocarcinoma.Involvement of mitochondria-caspase pathway in Hemoporfin-mediated cell death.Leptin OB3 peptide suppresses leptin-induced signaling and progression in ovarian cancer cells.Gonadotropins regulate N-cadherin-mediated human ovarian surface epithelial cell survival at both post-translational and transcriptional levels through a cyclic AMP/protein kinase A pathway.Luteinizing hormone compromises the in vivo anti-tumor effect of cisplatin on human epithelial ovarian cancer cells.Follicle-Stimulating Hormone Receptor Is Expressed by Most Ovarian Cancer Subtypes and Is a Safe and Effective Immunotherapeutic Target.Granulosa Cell-Specific Brca1 Loss Alone or Combined with Trp53 Haploinsufficiency and Transgenic FSH Expression Fails to Induce Ovarian Tumors.Dynamic alterations of the extracellular environment of ovarian surface epithelial cells in premalignant transformation, tumorigenicity, and metastasis.Estrogen combined with progesterone decreases cell proliferation and inhibits the expression of Bcl-2 via microRNA let-7a and miR-34b in ovarian cancer cells.Single nucleotide polymorphisms of follicle-stimulating hormone receptor are associated with ovarian cancer susceptibility.
P2860
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P2860
Ovarian epithelial tumor growth promotion by follicle-stimulating hormone and inhibition of the effect by luteinizing hormone.
description
2000 nî lūn-bûn
@nan
2000年の論文
@ja
2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
2000年论文
@zh
2000年论文
@zh-cn
name
Ovarian epithelial tumor growt ...... effect by luteinizing hormone.
@en
type
label
Ovarian epithelial tumor growt ...... effect by luteinizing hormone.
@en
prefLabel
Ovarian epithelial tumor growt ...... effect by luteinizing hormone.
@en
P2093
P356
P1433
P1476
Ovarian epithelial tumor growt ...... effect by luteinizing hormone.
@en
P2093
Lauchlan SC
P356
10.1006/GYNO.1999.5628
P407
P577
2000-01-01T00:00:00Z