Activated Microglia-Induced Deficits in Excitatory Synapses Through IL-1β: Implications for Cognitive Impairment in Sepsis.

Activated Microglia-Induced Deficits in Excitatory Synapses Through IL-1β: Implications for Cognitive Impairment in Sepsis.

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http://www.wikidata.org/entity/Q41725625
Spermine reverses lipopolysaccharide-induced memory deficit in mice.Cecal Ligation and Puncture Results in Long-Term Central Nervous System Myeloid Inflammation.Synapse-specific IL-1 receptor subunit reconfiguration augments vulnerability to IL-1β in the aged hippocampus.Resveratrol Protects against Sepsis-Associated Encephalopathy and Inhibits the NLRP3/IL-1β Axis in Microglia.Preclinical models of overwhelming sepsis implicate the neural system that encodes contextual fear memory.Age-Dependent Changes in the Activation and Regulation of Microglia.Brain Barrier Breakdown as a Cause and Consequence of Neuroinflammation in Sepsis.Vasopressin Impairment During Sepsis Is Associated with Hypothalamic Intrinsic Apoptotic Pathway and Microglial Activation.Inflammatory and Glutamatergic Homeostasis Are Involved in Successful Aging.Pharmacological Tools to Activate Microglia and their Possible use to Study Neural Network Patho-physiology.P2X7 Receptor Signaling Contributes to Sepsis-Associated Brain Dysfunction.Hypercapnia induces IL-1β overproduction via activation of NLRP3 inflammasome: implication in cognitive impairment in hypoxemic adult rats.Age-related cognitive impairment is associated with long-term neuroinflammation and oxidative stress in a mouse model of episodic systemic inflammation.Brain-Defective Insulin Signaling Is Associated to Late Cognitive Impairment in Post-Septic Mice.Neuroinflammation-Induced Downregulation of Hippocampacal Neuregulin 1-ErbB4 Signaling in the Parvalbumin Interneurons Might Contribute to Cognitive Impairment in a Mouse Model of Sepsis-Associated Encephalopathy.The Communication Between the Immune and Nervous Systems: The Role of IL-1β in Synaptopathies.Dimethyl fumarate attenuates reactive microglia and long-term memory deficits following systemic immune challenge.
P2860

Activated Microglia-Induced Deficits in Excitatory Synapses Through IL-1β: Implications for Cognitive Impairment in Sepsis.

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http://www.wikidata.org/entity/Q41725625
description
2014 nî lūn-bûn
@nan
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
2014年论文
@zh
2014年论文
@zh-cn
name
Activated Microglia-Induced De ...... ognitive Impairment in Sepsis.
@en
type
label
Activated Microglia-Induced De ...... ognitive Impairment in Sepsis.
@en
prefLabel
Activated Microglia-Induced De ...... ognitive Impairment in Sepsis.
@en
P1433
P1476
P2093
P2860
P2888
P304
P31
P356
P433
P478
P50
P577
P5875
P6179
P698
P921
P356
P1433
P1476
Activated Microglia-Induced De ...... ognitive Impairment in Sepsis.
@en
P2093
P2860
P2888
P304
P31
P356
P433
P478
P50
P577
P5875
P6179
P698
P921