Determinants of atherosclerosis susceptibility in the C3H and C57BL/6 mouse model: evidence for involvement of endothelial cells but not blood cells or cholesterol metabolism.
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Ribosomal protein L17, RpL17, is an inhibitor of vascular smooth muscle growth and carotid intima formationMapping and Congenic Dissection of Genetic Loci Contributing to Hyperglycemia and Dyslipidemia in Mice.Genetic linkage of hyperglycemia and dyslipidemia in an intercross between BALB/cJ and SM/J Apoe-deficient mouse strains.Variation in Type 2 Diabetes-Related Phenotypes among Apolipoprotein E-Deficient Mouse StrainsInfluence of phthalates on glucose homeostasis and atherosclerosis in hyperlipidemic micePET imaging detection of macrophages with a formyl peptide receptor antagonistIncreased production of 12/15 lipoxygenase eicosanoids accelerates monocyte/endothelial interactions in diabetic db/db mice.Atherosclerosis susceptibility Loci identified in an extremely atherosclerosis-resistant mouse strain.Genome scans for transmission ratio distortion regions in miceCharacterization of Bglu3, a mouse fasting glucose locus, and identification of Apcs as an underlying candidate gene.Characterization of Ath29, a major mouse atherosclerosis susceptibility locus, and identification of Rcn2 as a novel regulator of cytokine expressionHyperglycemia in apolipoprotein E-deficient mouse strains with different atherosclerosis susceptibility.Identification of Soat1 as a quantitative trait locus gene on mouse chromosome 1 contributing to hyperlipidemiaLipid retention in the arterial wall of two mouse strains with different atherosclerosis susceptibility.Neointimal formation in two apolipoprotein E-deficient mouse strains with different atherosclerosis susceptibility.Genes within the MHC region have a dramatic influence on radiation-enhanced atherosclerosis in miceMicroarray analysis of gene expression in mouse aorta reveals role of the calcium signaling pathway in control of atherosclerosis susceptibility.Oxidized phospholipids as triggers of inflammation in atherosclerosis.Quantitative trait locus analysis of neointimal formation in an intercross between C57BL/6 and C3H/HeJ apolipoprotein E-deficient mice.Quantitative trait locus analysis of atherosclerosis in an intercross between C57BL/6 and C3H mice carrying the mutant apolipoprotein E gene.Genetic linkage of hyperglycemia, body weight and serum amyloid-P in an intercross between C57BL/6 and C3H apolipoprotein E-deficient mice.Association of a Vcam1 mutation with atherosclerosis susceptibility in diet-induced models of atherosclerosis.Quantitative trait locus analysis of carotid atherosclerosis in an intercross between C57BL/6 and C3H apolipoprotein E-deficient mice.The atheroprotective effect of 17beta-estradiol depends on complex interactions in adaptive immunityFacilitated replacement of Kupffer cells expressing a paraoxonase-1 transgene is essential for ameliorating atherosclerosis in mice.Genetic modifiers of atherosclerosis in miceSpecific monocyte adhesion to endothelial cells induced by oxidized phospholipids involves activation of cPLA2 and lipoxygenase.Tissue-specific expression and regulation of sexually dimorphic genes in mice.Innate immunity and monocyte-macrophage activation in atherosclerosis.Identification of inflammatory gene modules based on variations of human endothelial cell responses to oxidized lipids.Animal models of calcific aortic valve diseaseAnimal models of atherosclerosis.Low-grade chronic inflammation in regions of the normal mouse arterial intima predisposed to atherosclerosisOxidized lipids: the two faces of vascular inflammation.Localization of atherosclerosis susceptibility loci to chromosomes 4 and 6 using the Ldlr knockout mouse modelGenetic-genomic replication to identify candidate mouse atherosclerosis modifier genes.Lack of Toll-like receptor 4 or myeloid differentiation factor 88 reduces atherosclerosis and alters plaque phenotype in mice deficient in apolipoprotein EExperimental models investigating the inflammatory basis of atherosclerosis.Mitochondrial genetic background modulates bioenergetics and susceptibility to acute cardiac volume overload.Influence of genetic background on genetically engineered mouse phenotypes.
P2860
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P2860
Determinants of atherosclerosis susceptibility in the C3H and C57BL/6 mouse model: evidence for involvement of endothelial cells but not blood cells or cholesterol metabolism.
description
2000 nî lūn-bûn
@nan
2000年の論文
@ja
2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
2000年论文
@zh
2000年论文
@zh-cn
name
Determinants of atherosclerosi ...... lls or cholesterol metabolism.
@en
type
label
Determinants of atherosclerosi ...... lls or cholesterol metabolism.
@en
prefLabel
Determinants of atherosclerosi ...... lls or cholesterol metabolism.
@en
P2093
P1433
P1476
Determinants of atherosclerosi ...... lls or cholesterol metabolism.
@en
P2093
P304
P356
10.1161/01.RES.86.10.1078
P577
2000-05-01T00:00:00Z