Nerve growth factor acts through cAMP-dependent protein kinase to increase the number of sodium channels in PC12 cells.
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Rheb interacts with Raf-1 kinase and may function to integrate growth factor- and protein kinase A-dependent signals.Leukemia inhibitory factor receptor signaling negatively modulates nerve growth factor-induced neurite outgrowth in PC12 cells and sympathetic neuronsSustained activation of extracellular signal-regulated kinase by nerve growth factor regulates c-fos protein stabilization and transactivation in PC12 cells.Modulation of skeletal muscle sodium channels by human myotonin protein kinase.The neuron as a dynamic electrogenic machine: modulation of sodium-channel expression as a basis for functional plasticity in neurons.Domains of E1A that bind p105Rb, p130, and p300 are required to block nerve growth factor-induced neurite growth in PC12 cells.Oxygen-sensing neurons in the central nervous system.Neuronal growth factor regulation of two different sodium channel types through distinct signal transduction pathways.NGF-induction of the metalloproteinase-transin/stromelysin in PC12 cells: involvement of multiple protein kinases.Signal transduction by nerve growth factor and fibroblast growth factor in PC12 cells requires a sequence of src and ras actions.The activity of cAMP-dependent protein kinase is required at a posttranslational level for induction of voltage-dependent sodium channels by peptide growth factors in PC12 cells.GTPase-deficient G alpha 16 and G alpha q induce PC12 cell differentiation and persistent activation of cJun NH2-terminal kinases.A branched signaling pathway for nerve growth factor is revealed by Src-, Ras-, and Raf-mediated gene inductionsMembrane-associated sodium channels and cytoplasmic precursors in glial cells. Immunocytochemical, electrophysiological, and pharmacological studies.Requirement of the adenovirus E1A transformation domain 1 for inhibition of PC12 cell neuronal differentiationGABAA-receptor-mediated conductance and action potential waveform in cutaneous and muscle afferent neurons of the adult rat: differential expression and response to nerve injuryDifferential effects of NGF and BDNF on axotomy-induced changes in GABA(A)-receptor-mediated conductance and sodium currents in cutaneous afferent neurons.Hypoxia and N6,O2'-dibutyryladenosine 3',5'-cyclic monophosphate, but not nerve growth factor, induce Na+ channels and hypertrophy in chromaffin-like arterial chemoreceptors.Nerve growth factor prevents the amblyopic effects of monocular deprivation.Separate cyclic AMP sensors for neuritogenesis, growth arrest, and survival of neuroendocrine cells.Voltage-gated sodium channel as a target for metastatic risk reduction with re-purposed drugs.Adenovirus E1A proteins stimulate inositol phospholipid metabolism in PC12 cells.Nerve growth factor enhances voltage-gated Na+ channel activity and Transwell migration in Mat-LyLu rat prostate cancer cell line.Primary motor neurons fail to up-regulate voltage-gated sodium channel Na(v)1.3/brain type III following axotomy resulting from spinal cord injury.Changes in protein phosphorylation in cultured neurons after exposure to methyl mercury.Neurotrophin-3 inhibits HCO absorption via a cAMP-dependent pathway in renal thick ascending limb.The signal transduction pathway underlying ion channel gene regulation by SP1-C-Jun interactions.Protein kinase C and cAMP-dependent protein kinase regulate the neuronal differentiation of immortalized raphe neurons.The cyclic adenosine monophosphate-dependent protein kinase (PKA) is required for the sustained activation of mitogen-activated kinases and gene expression by nerve growth factor.Differential regulation of chromogranin B and synapsin I gene promoter activity by cAMP and cAMP-dependent protein kinase.Sodium currents during differentiation in a human neuroblastoma cell line.Stimulation of neuropeptide Y gene expression by brain-derived neurotrophic factor requires both the phospholipase Cgamma and Shc binding sites on its receptor, TrkB.Nerve growth factor in combination with second messenger analogues causes neuronal differentiation of PC12 cells expressing a dominant inhibitory Ras protein without inducing activation of extracellular signal-regulated kinases.Nerve growth factor stimulates rapid metabolic responses in PC12 cells.The delayed depolarization in rat cutaneous afferent axons is reduced following nerve transection and ligation, but not crush: implications for injury-induced axonal Na+ channel reorganization.Role of cell-cell interactions in the developmental regulation of Ca2+-activated K+ currents in vertebrate neurons.Differential modulation of potassium currents by cAMP and its long-term and short-term effects: dunce and rutabaga mutants of Drosophila.
P2860
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P2860
Nerve growth factor acts through cAMP-dependent protein kinase to increase the number of sodium channels in PC12 cells.
description
1990 nî lūn-bûn
@nan
1990年の論文
@ja
1990年論文
@yue
1990年論文
@zh-hant
1990年論文
@zh-hk
1990年論文
@zh-mo
1990年論文
@zh-tw
1990年论文
@wuu
1990年论文
@zh
1990年论文
@zh-cn
name
Nerve growth factor acts throu ...... sodium channels in PC12 cells.
@en
type
label
Nerve growth factor acts throu ...... sodium channels in PC12 cells.
@en
prefLabel
Nerve growth factor acts throu ...... sodium channels in PC12 cells.
@en
P2093
P1433
P1476
Nerve growth factor acts throu ...... sodium channels in PC12 cells.
@en
P2093
P304
P356
10.1016/0896-6273(90)90048-K
P407
P577
1990-03-01T00:00:00Z