Do we still need dipyridamole? - Wikidata - awgldk - TriplyDB

Do we still need dipyridamole?

Do we still need dipyridamole?

http://www.wikidata.org/entity/Q41760001

about

Design of the Dialysis Access Consortium (DAC) Aggrenox Prevention Of Access Stenosis Trial A critically severe gingival bleeding following non-surgical periodontal treatment in patients medicated with anti-platelet.

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Do we still need dipyridamole?

http://www.wikidata.org/entity/Q41760001

description

1998 nî lūn-bûn

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1998年の論文

@ja

1998年論文

@yue

1998年論文

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1998年論文

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1998年論文

@zh-mo

1998年論文

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1998年论文

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1998年论文

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1998年论文

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Do we still need dipyridamole?

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Do we still need dipyridamole?

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Do we still need dipyridamole?

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J.1365-2125.1998.T01-1-00677.X

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British Journal of Clinical Pharmacology

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Do we still need dipyridamole?

@en

P2093

C R Gibbs

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G Y Lip

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P2860

GISSI-3: effects of lisinopril and transdermal glyceryl trinitrate singly and together on 6-week mortality and ventricular function after acute myocardial infarction. Gruppo Italiano per lo Studio della Sopravvivenza nell'infarto Miocardico

The safety of intravenous dipyridamole thallium myocardial perfusion imaging. Intravenous Dipyridamole Thallium Imaging Study Group

Collaborative overview of randomised trials of antiplatelet therapy--I: Prevention of death, myocardial infarction, and stroke by prolonged antiplatelet therapy in various categories of patients. Antiplatelet Trialists' Collaboration

Quantitative comparison of thallium-201 scintigraphy after exercise and dipyridamole in coronary artery disease.

Platelet and fibrinogen consumption in man.

Effect of dipyridamole and aspirin on late vein-graft patency after coronary bypass operations.

Effect of intravenous nitrates on mortality in acute myocardial infarction: an overview of the randomised trials.

Effects of intravenous magnesium in suspected acute myocardial infarction: overview of randomised trials

Dipyridamole-echocardiography test: historical background and physiologic basis.

Dipyridamole and other phosphodiesterase inhibitors act as antithrombotic agents by potentiating endogenous prostacyclin.

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323-328

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scholarly article

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10.1046/J.1365-2125.1998.T01-1-00677.X

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227893334

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