about
Functions of Ubiquitin and SUMO in DNA Replication and Replication StressA role for H2B ubiquitylation in DNA replicationA novel role for the Pol I transcription factor UBTF in maintaining genome stability through the regulation of highly transcribed Pol II genes.Histone H3K56 acetylation, CAF1, and Rtt106 coordinate nucleosome assembly and stability of advancing replication forksChromatin replication and epigenome maintenance.The chromatin assembly factor 1 promotes Rad51-dependent template switches at replication forks by counteracting D-loop disassembly by the RecQ-type helicase Rqh1Rescue from replication stress during mitosis.H2B mono-ubiquitylation facilitates fork stalling and recovery during replication stress by coordinating Rad53 activation and chromatin assembly.Replicating damaged DNA in eukaryotes.Histone H2B mono-ubiquitylation maintains genomic integrity at stalled replication forks.Regulation of Replication Fork Advance and Stability by Nucleosome Assembly.Monoubiquitylation of histone H2B contributes to the bypass of DNA damage during and after DNA replicationUbiquitin family modifications and template switching.Replication fork dynamics and the DNA damage response.Nucleosome assembly and genome integrity: The fork is the link.Class I HDACs Affect DNA Replication, Repair, and Chromatin Structure: Implications for Cancer Therapy.Homologous recombination as a replication fork escort: fork-protection and recovery.Histone availability as a strategy to control gene expressionRad51 replication fork recruitment is required for DNA damage tolerance.Histone dosage regulates DNA damage sensitivity in a checkpoint-independent manner by the homologous recombination pathway.Defective histone supply causes condensin-dependent chromatin alterations, SAC activation and chromosome decatenation impairment.Genetic instability is prevented by Mrc1-dependent spatio-temporal separation of replicative and repair activities of homologous recombination: homologous recombination tolerates replicative stress by Mrc1-regulated replication and repair activitiesHigh-resolution breakpoint analysis provides evidence for the sequence-directed nature of genome rearrangements in hereditary disorders.EZH2 promotes degradation of stalled replication forks by recruiting MUS81 through histone H3 trimethylation.Histone depletion prevents telomere fusions in pre-senescent cells.
P2860
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P2860
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
2009年论文
@zh
2009年论文
@zh-cn
name
Chromatin assembly controls replication fork stability.
@en
type
label
Chromatin assembly controls replication fork stability.
@en
prefLabel
Chromatin assembly controls replication fork stability.
@en
P2860
P356
P1433
P1476
Chromatin assembly controls replication fork stability
@en
P2093
Marta Clemente-Ruiz
P2860
P304
P356
10.1038/EMBOR.2009.67
P50
P577
2009-05-22T00:00:00Z