A novel subset of mouse NKT cells bearing the IL-17 receptor B responds to IL-25 and contributes to airway hyperreactivity. - Wikidata - awgldk - TriplyDB

A novel subset of mouse NKT cells bearing the IL-17 receptor B responds to IL-25 and contributes to airway hyperreactivity.

A novel subset of mouse NKT cells bearing the IL-17 receptor B responds to IL-25 and contributes to airway hyperreactivity.

http://www.wikidata.org/entity/Q41770910

about

The transcription factor E4BP4 regulates the production of IL-10 and IL-13 in CD4+ T cells Blocking IL-25 signalling protects against gut inflammation in a type-2 model of colitis by suppressing nuocyte and NKT derived IL-13 IL25 elicits a multipotent progenitor cell population that promotes T(H)2 cytokine responses Interleukin (IL)-25: Pleiotropic roles in asthma The Extended Family of CD1d-Restricted NKT Cells: Sifting through a Mixed Bag of TCRs, Antigens, and Functions Fine tuning a well-oiled machine: Influence of NK1.1 and NKG2D on NKT cell development and function Stages versus subsets: Invariant Natural Killer T cell lineage differentiation Cytokine-induced cytokine production by conventional and innate lymphoid cells Innate lymphoid cells in the airways Transcriptional regulation of the NKT cell lineage Concerted activity of IgG1 antibodies and IL-4/IL-25-dependent effector cells trap helminth larvae in the tissues following vaccination with defined secreted antigens, providing sterile immunity to challenge infection Mechanisms for Glycolipid Antigen-Driven Cytokine Polarization by V 14i NKT Cells Diacylglycerol Kinases in T Cell Tolerance and Effector Function A novel small compound SH-2251 suppresses Th2 cell-dependent airway inflammation through selective modulation of chromatin status at the Il5 gene locus Raising the roof: the preferential pharmacological stimulation of Th1 and th2 responses mediated by NKT cells.T cell factor 1 is required for group 2 innate lymphoid cell generation.Regulation of IL-9 expression by IL-25 signaling.Induction of airway hyperreactivity by IL-25 is dependent on a subset of invariant NKT cells expressing IL-17RB.PD-L1 and PD-L2 modulate airway inflammation and iNKT-cell-dependent airway hyperreactivity in opposing directions.A CD1d-dependent antagonist inhibits the activation of invariant NKT cells and prevents development of allergen-induced airway hyperreactivity Mechanisms of innate lymphoid cell and natural killer T cell activation during mucosal inflammation.Early local immune defences in the respiratory tract Runt-related transcription factor 3 is involved in the altered phenotype and function in ThPok-deficient invariant natural killer T cells Cytokine dependent and independent iNKT cell activation Chronic inflammation and asthma.ID'ing innate and innate-like lymphoid cells Development and function of invariant natural killer T cells producing T(h)2- and T(h)17-cytokines Increased Th2-like Invariant Natural Killer T cells in Peripheral Blood From Patients With Asthma.Influenza infection in suckling mice expands an NKT cell subset that protects against airway hyperreactivity.Tissue-Specific Distribution of iNKT Cells Impacts Their Cytokine Response.Development of invariant natural killer T cells.Cytokine/anti-cytokine therapy - novel treatments for asthma?IL-1β suppresses innate IL-25 and IL-33 production and maintains helminth chronicity.Mucosal immune responses following intestinal nematode infection PLZF induces an intravascular surveillance program mediated by long-lived LFA-1-ICAM-1 interactions.CD1d Expression and Invariant NKT Cell Responses in Herpesvirus Infections β-Catenin is required for the differentiation of iNKT2 and iNKT17 cells that augment IL-25-dependent lung inflammation.Cutting Edge: IL-25 Targets Dendritic Cells To Attract IL-9-Producing T Cells in Acute Allergic Lung Inflammation.Asthma as a chronic disease of the innate and adaptive immune systems responding to viruses and allergens Suppression of inflammation in ulcerative colitis by interferon-β-1a is accompanied by inhibition of IL-13 production.

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P2860

A novel subset of mouse NKT cells bearing the IL-17 receptor B responds to IL-25 and contributes to airway hyperreactivity.

http://www.wikidata.org/entity/Q41770910

description

2008 nî lūn-bûn

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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2008年论文

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2008年论文

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name

A novel subset of mouse NKT ce ...... tes to airway hyperreactivity.

@en

type

label

A novel subset of mouse NKT ce ...... tes to airway hyperreactivity.

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prefLabel

A novel subset of mouse NKT ce ...... tes to airway hyperreactivity.

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P1433

Journal of Experimental Medicine

P1476

A novel subset of mouse NKT ce ...... tes to airway hyperreactivity.

@en

P2093

Asuka Terashima

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Chiaki Iwamura

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Etsuko Sekine

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Hiroshi Nakajima

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Hiroshi Watarai

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Koji Hase

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Masaru Taniguchi

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Ryusuke Nakagawa

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Sayo Inoue

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P2860

Identification of an interleukin (IL)-25-dependent cell population that provides IL-4, IL-5, and IL-13 at the onset of helminth expulsion

Interleukin-17 family and IL-17 receptors

Mast cells produce interleukin-25 upon Fc epsilon RI-mediated activation

Transgenic overexpression of human IL-17E results in eosinophilia, B-lymphocyte hyperplasia, and altered antibody production

Forced expression of murine IL-17E induces growth retardation, jaundice, a Th2-biased response, and multiorgan inflammation in mice

IL-25 induces IL-4, IL-5, and IL-13 and Th2-associated pathologies in vivo

IL-25 enhances allergic airway inflammation by amplifying a TH2 cell-dependent pathway in mice

Interleukin-13: central mediator of allergic asthma

IL-17E upregulates the expression of proinflammatory cytokines in lung fibroblasts

New IL-17 family members promote Th1 or Th2 responses in the lung: in vivo function of the novel cytokine IL-25

P304

2727-2733

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10.1084/JEM.20080698

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12

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205

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Toshinori Nakayama

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2008-11-17T00:00:00Z

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23479566

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19015310

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2585837

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