Fsr-independent production of protease(s) may explain the lack of attenuation of an Enterococcus faecalis fsr mutant versus a gelE-sprE mutant in induction of endocarditis.
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Endocarditis and biofilm-associated pili of Enterococcus faecalisVancomycin-resistant Enterococcus faecalis endocarditis: linezolid failure and strain characterization of virulence factorsImportance of the collagen adhesin ace in pathogenesis and protection against Enterococcus faecalis experimental endocarditis.Bicarbonate enhances expression of the endocarditis and biofilm associated pilus locus, ebpR-ebpABC, in Enterococcus faecalis.Gelatinase contributes to the pathogenesis of endocarditis caused by Enterococcus faecalis.Diversity of the fsr-gelE region of the Enterococcus faecalis genome but conservation in strains with partial deletions of the fsr operon.Full activation of Enterococcus faecalis gelatinase by a C-terminal proteolytic cleavage.Drosophila host model reveals new enterococcus faecalis quorum-sensing associated virulence factors.The Enterococcus faecium enterococcal biofilm regulator, EbrB, regulates the esp operon and is implicated in biofilm formation and intestinal colonization.Enterococcal endocarditis: can we win the war?Multiple roles for Enterococcus faecalis glycosyltransferases in biofilm-associated antibiotic resistance, cell envelope integrity, and conjugative transfer.Genomic diversification of enterococci in hosts: the role of the mobilome.The rise of the Enterococcus: beyond vancomycin resistance.Large scale variation in Enterococcus faecalis illustrated by the genome analysis of strain OG1RF.Importance of the ebp (endocarditis- and biofilm-associated pilus) locus in the pathogenesis of Enterococcus faecalis ascending urinary tract infection.A fratricidal mechanism is responsible for eDNA release and contributes to biofilm development of Enterococcus faecalis.Targeting pili in enterococcal pathogenesis.Enterococcus infection biology: lessons from invertebrate host models.The Fsr quorum-sensing system of Enterococcus faecalis modulates surface display of the collagen-binding MSCRAMM Ace through regulation of gelE.Model systems for the study of Enterococcal colonization and infection.Efficacy of Telavancin Alone and in Combination with Ampicillin in a Rat Model of Enterococcus faecalis Endocarditis.Decreased virulence of a gls24 mutant of Enterococcus faecalis OG1RF in an experimental endocarditis model.Lack of correlation of gelatinase production and biofilm formation in a large collection of Enterococcus faecalis isolates.Comparison of OG1RF and an isogenic fsrB deletion mutant by transcriptional analysis: the Fsr system of Enterococcus faecalis is more than the activator of gelatinase and serine protease.Processing of the major autolysin of E. faecalis, AtlA, by the zinc-metalloprotease, GelE, impacts AtlA septal localization and cell separation.Introduction of a Qualified Presumption of Safety (QPS) approach for assessment of selected microorganisms referred to EFSA - Opinion of the Scientific CommitteePhenotypic and genotypic study of gelatinase and beta-haemolysis activities in faecal enterococci of poultry in Portugal.
P2860
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P2860
Fsr-independent production of protease(s) may explain the lack of attenuation of an Enterococcus faecalis fsr mutant versus a gelE-sprE mutant in induction of endocarditis.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
2005年论文
@zh
2005年论文
@zh-cn
name
Fsr-independent production of ...... in induction of endocarditis.
@en
type
label
Fsr-independent production of ...... in induction of endocarditis.
@en
prefLabel
Fsr-independent production of ...... in induction of endocarditis.
@en
P2093
P2860
P1476
Fsr-independent production of ...... in induction of endocarditis.
@en
P2093
Barbara E Murray
Kavindra V Singh
Sreedhar R Nallapareddy
P2860
P304
P356
10.1128/IAI.73.8.4888-4894.2005
P407
P577
2005-08-01T00:00:00Z