about
Heat shock proteins and hormesis in the diagnosis and treatment of neurodegenerative diseasesThe Janus face of the heme oxygenase/biliverdin reductase system in Alzheimer disease: it's time for reconciliationIn vivo induction of heat shock proteins in the substantia nigra following L-DOPA administration is associated with increased activity of mitochondrial complex I and nitrosative stress in rats: regulation by glutathione redox stateSex differences in brain proteomes of neuron-specific STAT3-null mice after cerebral ischemia/reperfusionbeta-Carotene and cigarette smoke condensate regulate heme oxygenase-1 and its repressor factor Bach1: relationship with cell growthLack of p53 affects the expression of several brain mitochondrial proteins: insights from proteomics into important pathways regulated by p53.Biliverdin reductase--a protein levels and activity in the brains of subjects with Alzheimer disease and mild cognitive impairment.Association between frontal cortex oxidative damage and beta-amyloid as a function of age in Down syndrome.Heme oxygenase and its products in the nervous system.Heme oxygenase-1 posttranslational modifications in the brain of subjects with Alzheimer disease and mild cognitive impairment.Oxidative stress and cellular stress response in diabetic nephropathy.Bach1 overexpression in Down syndrome correlates with the alteration of the HO-1/BVR-a system: insights for transition to Alzheimer's disease.Heme oxygenase and cyclooxygenase in the central nervous system: a functional interplay.Bilirubin: an endogenous scavenger of nitric oxide and reactive nitrogen species.Redox regulation of cellular stress response in aging and neurodegenerative disorders: role of vitagenes.Oxidatively-modified and glycated proteins as candidate pro-inflammatory toxins in uremia and dialysis patients.Cellular stress response: a novel target for chemoprevention and nutritional neuroprotection in aging, neurodegenerative disorders and longevity.Curcumin and the cellular stress response in free radical-related diseases.The role of antioxidant supplement in immune system, neoplastic, and neurodegenerative disorders: a point of view for an assessment of the risk/benefit profileVitagenes, dietary antioxidants and neuroprotection in neurodegenerative diseases.The heme oxygenase/biliverdin reductase pathway in drug research and development.Impairment of proteostasis network in Down syndrome prior to the development of Alzheimer's disease neuropathology: redox proteomics analysis of human brain.Roles of nitric oxide, carbon monoxide, and hydrogen sulfide in the regulation of the hypothalamic-pituitary-adrenal axis.The hormetic role of dietary antioxidants in free radical-related diseases.Pharmacologists and Alzheimer disease therapy: to boldly go where no scientist has gone before.Natural substances and Alzheimer's disease: from preclinical studies to evidence based medicine.Ferulic acid: pharmacological and toxicological aspects.Key factors which concur to the correct therapeutic evaluation of herbal products in free radical-induced diseases.Panax ginseng and Panax quinquefolius: From pharmacology to toxicology.Ferulic Acid Regulates the Nrf2/Heme Oxygenase-1 System and Counteracts Trimethyltin-Induced Neuronal Damage in the Human Neuroblastoma Cell Line SH-SY5Y.The contribution of transgenic and nontransgenic animal models in Alzheimer's disease drug research and development.Bilirubin and brain: A pharmacological approach.KV7 channels in the human detrusor: channel modulator effects and gene and protein expression.Heme oxygenase-derived carbon monoxide modulates gonadotropin-releasing hormone release in immortalized hypothalamic neurons.Heme oxygenase expression and activity in immortalized hypothalamic neurons GT1-7.Caspase-3 inhibits the growth of breast cancer cells independent of protease activity.Lack of p53 decreases basal oxidative stress levels in the brain through upregulation of thioredoxin-1, biliverdin reductase-A, manganese superoxide dismutase, and nuclear factor kappa-B.Redox proteomics analyses of the influence of co-expression of wild-type or mutated LRRK2 and Tau on C. elegans protein expression and oxidative modification: relevance to Parkinson diseasePreclinical and clinical issues in Alzheimer's disease drug research and development.Evidence that carbon monoxide stimulates prostaglandin endoperoxide synthase activity in rat hypothalamic explants and in primary cultures of rat hypothalamic astrocytes.
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