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Network pharmacology exploration reveals endothelial inflammation as a common mechanism for stroke and coronary artery disease treatment of Danhong injection.Irisin Alleviates Advanced Glycation End Products-Induced Inflammation and Endothelial Dysfunction via Inhibiting ROS-NLRP3 Inflammasome Signaling.Dysregulation of Calpain Proteolytic Systems Underlies Degenerative Vascular Disorders.Nicotine promotes atherosclerosis via ROS-NLRP3-mediated endothelial cell pyroptosis.NLRP3: A Novel Mediator in Cardiovascular Disease.Age and Age-Related Diseases: Role of Inflammation Triggers and Cytokines.Liquefaction of the Brain following Stroke Shares a Similar Molecular and Morphological Profile with Atherosclerosis and Mediates Secondary Neurodegeneration in an Osteopontin-Dependent Mechanism
P2860
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P2860
description
2017 nî lūn-bûn
@nan
2017年の論文
@ja
2017年論文
@yue
2017年論文
@zh-hant
2017年論文
@zh-hk
2017年論文
@zh-mo
2017年論文
@zh-tw
2017年论文
@wuu
2017年论文
@zh
2017年论文
@zh-cn
name
Role of NLRP3 Inflammasomes in Atherosclerosis.
@en
type
label
Role of NLRP3 Inflammasomes in Atherosclerosis.
@en
prefLabel
Role of NLRP3 Inflammasomes in Atherosclerosis.
@en
P2860
P921
P356
P1476
Role of NLRP3 Inflammasomes in Atherosclerosis.
@en
P2093
Masafumi Takahashi
Tadayoshi Karasawa
P2860
P304
P356
10.5551/JAT.RV17001
P577
2017-03-04T00:00:00Z
2017-05-01T00:00:00Z