DJ-1 deficiency in astrocytes selectively enhances mitochondrial Complex I inhibitor-induced neurotoxicity.
about
Gene-environment interactions in Parkinson's disease: specific evidence in humans and mammalian modelsMitochondrial Redox Dysfunction and Environmental ExposuresSerotonin 1A Receptors on Astrocytes as a Potential Target for the Treatment of Parkinson's DiseaseDJ-1-dependent regulation of oxidative stress in the retinal pigment epithelium (RPE)Behavioral and neurotransmitter abnormalities in mice deficient for Parkin, DJ-1 and superoxide dismutase.DJ-1 knock-down impairs astrocyte mitochondrial function.Viability assays for cells in culture.Lipoxin A4 Activates Nrf2 Pathway and Ameliorates Cell Damage in Cultured Cortical Astrocytes Exposed to Oxygen-Glucose Deprivation/Reperfusion Insults.Neocortex and allocortex respond differentially to cellular stress in vitro and aging in vivoAstrocytes in Parkinson's disease and DJ-1The role of astrocyte mitochondria in differential regional susceptibility to environmental neurotoxicants: tools for understanding neurodegenerationLife-time expression of the proteins peroxiredoxin, beta-synuclein, PARK7/DJ-1, and stathmin in the primary visual and primary somatosensory cortices in rats.The Polg Mutator Phenotype Does Not Cause Dopaminergic Neurodegeneration in DJ-1-Deficient Mice.Neuron-astrocyte interactions in neurodegenerative diseases: Role of neuroinflammationIdentification of chemicals that mimic transcriptional changes associated with autism, brain aging and neurodegeneration.Neuroprotective function of DJ-1 in Parkinson's disease.Surprising behavioral and neurochemical enhancements in mice with combined mutations linked to Parkinson's disease.Mitochondria, calcium, and endoplasmic reticulum stress in Parkinson's disease.αSynuclein and Mitochondrial Dysfunction: A Pathogenic Partnership in Parkinson's Disease?Astrocytes Surviving Severe Stress Can Still Protect Neighboring Neurons from Proteotoxic Injury.Knocking out DJ-1 attenuates astrocytes neuroprotection against 6-hydroxydopamine toxicity.Metabolic Dysfunction in Parkinson's Disease: Bioenergetics, Redox Homeostasis and Central Carbon Metabolism.The Role of Astrocyte Dysfunction in Parkinson's Disease Pathogenesis.Post-Translational Oxidative Modifications of Mitochondrial Complex I (NADH: Ubiquinone Oxidoreductase): Implications for Pathogenesis and Therapeutics in Human Diseases.DJ-1 expression modulates astrocyte-mediated protection against neuronal oxidative stress.Diversity of astroglial responses across human neurodegenerative disorders and brain aging.Lipoxin A4 inhibits 5-lipoxygenase translocation and leukotrienes biosynthesis to exert a neuroprotective effect in cerebral ischemia/reperfusion injury.DJ-1 modulates mitochondrial response to oxidative stress: clues from a novel diagnosis of PARK7.Astrocyte-specific DJ-1 overexpression protects against rotenone-induced neurotoxicity in a rat model of Parkinson's disease.Astroglial DJ-1 over-expression up-regulates proteins involved in redox regulation and is neuroprotective in vivo.Astrocytes, Microglia, and Parkinson's Disease.
P2860
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P2860
DJ-1 deficiency in astrocytes selectively enhances mitochondrial Complex I inhibitor-induced neurotoxicity.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
DJ-1 deficiency in astrocytes ...... hibitor-induced neurotoxicity.
@en
type
label
DJ-1 deficiency in astrocytes ...... hibitor-induced neurotoxicity.
@en
prefLabel
DJ-1 deficiency in astrocytes ...... hibitor-induced neurotoxicity.
@en
P2860
P1476
DJ-1 deficiency in astrocytes ...... hibitor-induced neurotoxicity.
@en
P2093
David A Hinkle
Steven J Mullett
P2860
P304
P356
10.1111/J.1471-4159.2011.07175.X
P407
P577
2011-01-28T00:00:00Z