Oxidative DNA damage is instrumental in hyperreplication stress-induced inviability of Escherichia coli.
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DNA Replication Control Is Linked to Genomic Positioning of Control Regions in Escherichia coli.Identification of β Clamp-DNA Interaction Regions That Impair the Ability of E. coli to Tolerate Specific Classes of DNA Damage.Re-wiring of energy metabolism promotes viability during hyperreplication stress in E. coli.Bactericidal Antibiotics Induce Toxic Metabolic Perturbations that Lead to Cellular DamageInfluenza infection induces host DNA damage and dynamic DNA damage responses during tissue regeneration.Escherichia coli DNA ligase B may mitigate damage from oxidative stress.The Escherichia coli Cryptic Prophage Protein YfdR Binds to DnaA and Initiation of Chromosomal Replication Is Inhibited by Overexpression of the Gene Cluster yfdQ-yfdR-yfdS-yfdTCountermeasures to survive excessive chromosome replication in Escherichia coli.The nucleoid occlusion factor Noc controls DNA replication initiation in Staphylococcus aureus.SeqA structures behind Escherichia coli replication forks affect replication elongation and restart mechanisms.Maladaptive DNA repair is the ultimate contributor to the death of trimethoprim-treated cells under aerobic and anaerobic conditions.Lethality of MalE-LacZ hybrid protein shares mechanistic attributes with oxidative component of antibiotic lethality.Identification of bacterial invasion in necrotizing enterocolitis specimens using fluorescent in situ hybridization.Targeting the Bacterial Orisome in the Search for New Antibiotics.The DnaA Cycle in Escherichia coli: Activation, Function and Inactivation of the Initiator Protein.Oxidation of dCTP contributes to antibiotic lethality in stationary-phase mycobacteria.Insufficient levels of the nrdAB-encoded ribonucleotide reductase underlie the severe growth defect of the Δhda E. coli strain.Improved measurements of scant hydrogen peroxide enable experiments that define its threshold of toxicity for Escherichia coli.The DnaA Tale.DnaA and LexA Proteins Regulate Transcription of the uvrB Gene in Escherichia coli: The Role of DnaA in the Control of the SOS Regulon.Iron chelation increases the tolerance of Escherichia coli to hyper-replication stress.Chromosome replication as a measure of bacterial growth rate during Escherichia coli infection in the mouse peritonitis model
P2860
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P2860
Oxidative DNA damage is instrumental in hyperreplication stress-induced inviability of Escherichia coli.
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2014 nî lūn-bûn
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2014年の論文
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2014年論文
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2014年論文
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2014年論文
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2014年論文
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2014年論文
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2014年论文
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name
Oxidative DNA damage is instru ...... viability of Escherichia coli.
@en
type
label
Oxidative DNA damage is instru ...... viability of Escherichia coli.
@en
prefLabel
Oxidative DNA damage is instru ...... viability of Escherichia coli.
@en
P2860
P50
P356
P1476
Oxidative DNA damage is instru ...... nviability of Escherichia coli
@en
P2093
Belén Mendoza-Chamizo
Louise Bjørn
P2860
P304
13228-13241
P356
10.1093/NAR/GKU1149
P577
2014-11-11T00:00:00Z