On the role of the H-2 histocompatibility complex in determining the specificity of cytotoxic effector cells sensitized against syngeneic trinitrophenyl-modified targets.
about
Absence of H-2 antigens capable of reacting with cytotoxic T cells on a teratoma line expressing a T/t locus antigenInhibition of cell-mediated cytolysis of trinitrophenyl-derivatized target cells by alloantisera directed to the products of the K and D loci of the H-2 complex.Histocompatibility antigens controlled by the I region of the murine H-2 complex. II. K/D region compatibility is not required for I-region cell-mediated lymphocytotoxicityJoint recognition by cytotoxic T cells of inactivated Sendai virus and products of the major histocompatibility complexCytotoxic T cells distinguish between trinitrophenyl- and dinitrophenyl-modified syngeneic cells.Specificity of cytotoxic effector cells directed against trinitrobenzene sulfonate-modified syngeneic cells. Failure to recognize cell surface-bound trinitrophenyl dextran.H-2 effects on cell-cell interactions in the response to single non-H-2 alloantigens. I. Donor H-2D region control of H-7.1-immunogenicity and lack of restriction in vivo.T-cell-mediated cytotoxic immune responses to F9 teratocarcinoma cells: cytolytic effector T cells lyse H-2-negative F9 cells and syngeneic spermatogonia.H-2-restricted cytotoxic effectors generated in vitro by the addition of trinitrophenyl-conjugated soluble proteinsH-2-linked genetic control of murine T-cell-mediated lympholysis to autologous cells modified with low concentrations of trinitrobenzene sulfonateRegulation of T-cell-mediated lympholysis by the murine major histocompatibility complex. I. Preferential in vitro responses to trinitrophenyl-modified self K- and D-coded gene products in parental and F1 hybrid mouse strainsSelective turnover and shedding of H-2K and H-2D antigens is controlled by the major histocompatibility complex. Implications for H-2-restricted recognition.Major histocompatibility complex restriction of soluble helper molecules in T cell responses to altered self.The Qa2 subregion controls the expression of two antigens recognized by H-2-unrestricted cytotoxic T cells.Non-H-2 restriction of expression of passively transferred delayed sensitivity.H-2L-restricted recognition of viral antigens In the H-2d haplotype, anti-vesicular stomatitis virus cytotoxic T cells are restricted solely by H-2L.Trinitrophenyl modification of H-2k and H-2b spleen cells results in enhanced serological detection of Kk-like determinants.Specific suppression of allograft rejection by trinitrophenyl (TNP)-induced suppressor cells in recipients treated with TNP-haptenated donor alloantigensCell-mediated lympholysis of trinitrophenyl-modified autologous lymphocytes. Confirmation of genetic control of response to trinitrophenyl-modified H-2 antigens by the use of anti-H-2 and anti-Ia antibodiesCell-mediated lympholysis of N-(3-nitro-4-hydroxy-5-iodophenylacetyl)-beta-anaylglycylglycyl-modified autologous lymphocytes. Effector cell specificity to modified cell surface components controlled by the H-2K and H-2D serological regions of the muH-2 compatibility requirement for virus-specific T-cell-mediated cytolysis. The H-2K structure involved is coded by a single cistron defined by H-2Kb mutant miceParticipation of the H-2 antigens of tumor cells in their lysis by syngeneic T cells.Histocompatibility antigens controlled by the I region of the murine H-2 complex. I. Mapping of H-2A and H-2C loci.T-cell-deficient mice produce more antihapten antibodies against syngeneic than against allogeneic erythrocyte conjugatesCross-reactive lysis of trinitrophenyl (TNP)-derivatized H-2 incompatible target cells by cytolytic T lymphocytes generated against syngeneic TNP spleen cellsMajor histocompatibility complex products restrict the adherence of cytolytic T lymphocytes to minor histocompatibility antigens or to trinitrophenyl determinants on schistosomula of Schistosoma mansoni.In vitro sensitization of human T lymphocytes to hapten (TNP)-conjugated and non-treated autologous cells is restricted by self-HLA-D.Cell membrane antigens recognized by anti-viral and anti-trinitrophenyl cytotoxic T lymphocytes.Use of hapten-altered self moieties to probe the cell-mediated lympholytic response and immunotolerance interface.Induction of immunity and suppression by haptenated self structures.In vivo generation and regulation of killer T-cells directed towards hapten-coupled syngeneic cells.Immune response to haptenated syngeneic and allogeneic lymphocytes. I. Restrictions for B cell activation by haptenated lymphocytes and specificity of the antibodies generated.Virus and trinitrophenol hapten-specific T-cell-mediated cytotoxicity against H-2 incompatible target cells.Cell surface structures required for B-cell activation with haptenated syngeneic cells.Comparison between the specificity of primary and secondary killer cells against alloantigens and hapten-modified syngeneic lymphoid cells.
P2860
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P2860
On the role of the H-2 histocompatibility complex in determining the specificity of cytotoxic effector cells sensitized against syngeneic trinitrophenyl-modified targets.
description
1975 nî lūn-bûn
@nan
1975年の論文
@ja
1975年論文
@yue
1975年論文
@zh-hant
1975年論文
@zh-hk
1975年論文
@zh-mo
1975年論文
@zh-tw
1975年论文
@wuu
1975年论文
@zh
1975年论文
@zh-cn
name
On the role of the H-2 histoco ...... initrophenyl-modified targets.
@en
type
label
On the role of the H-2 histoco ...... initrophenyl-modified targets.
@en
prefLabel
On the role of the H-2 histoco ...... initrophenyl-modified targets.
@en
P2860
P356
P1476
On the role of the H-2 histoco ...... initrophenyl-modified targets.
@en
P2093
P2860
P304
P356
10.1084/JEM.142.2.403
P407
P577
1975-08-01T00:00:00Z