Double dissociation of the requirement for GluN2B- and GluN2A-containing NMDA receptors in the destabilization and restabilization of a reconsolidating memory.
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Pharmacology of cognitive enhancers for exposure-based therapy of fear, anxiety and trauma-related disordersNoradrenergic regulation of fear and drug-associated memory reconsolidationCholinergic manipulations bidirectionally regulate object memory destabilization.Late Arc/Arg3.1 expression in the basolateral amygdala is essential for persistence of newly-acquired and reactivated contextual fear memories.Amygdala Dopamine Receptors Are Required for the Destabilization of a Reconsolidating Appetitive Memory(1,2).Dopamine D1-like receptor signalling in the hippocampus and amygdala modulates the acquisition of contextual fear conditioningRetrieval cues that trigger reconsolidation of associative fear memory are not necessarily an exact replica of the original learning experienceContribution of an SFK-Mediated Signaling Pathway in the Dorsal Hippocampus to Cocaine-Memory Reconsolidation in Rats.Glucocorticoid receptor antagonism disrupts the reconsolidation of social reward-related memories in ratsXenon impairs reconsolidation of fear memories in a rat model of post-traumatic stress disorder (PTSD)Memories reactivated under ketamine are subsequently stronger: A potential pre-clinical behavioral model of psychosisReconsolidation allows fear memory to be updated to a less aversive level through the incorporation of appetitive information.Assessing the translational feasibility of pharmacological drug memory reconsolidation blockade with memantine in quitting smokers.An Update on Memory Reconsolidation Updating.Frequency-specific stimulations induce reconsolidation of long-term potentiation in freely moving rats.Increasing the GluN2A/GluN2B Ratio in Neurons of the Mouse Basal and Lateral Amygdala Inhibits the Modification of an Existing Fear Memory Trace.Hypothesizing the body's genius to trigger and self-organize its healing: 25 years using a standardized neurophysics therapy.Involvement of amygdalar protein kinase A, but not calcium/calmodulin-dependent protein kinase II, in the reconsolidation of cocaine-related contextual memories in rats.Mechanisms governing the reactivation-dependent destabilization of memories and their role in extinction.Reconsolidation and extinction are dissociable and mutually exclusive processes: behavioral and molecular evidence.Co-activation of NR2A and NR2B subunits induces resistance to fear extinction.Memory reconsolidation in aversive and appetitive settings.Metabotropic glutamate receptor 5 as a potential target for smoking cessation.Memory Reconsolidation.Inhibition and enhancement of contextual fear memory destabilization.Inhibition of protein synthesis but not β-adrenergic receptors blocks reconsolidation of a cocaine-associated cue memory.Disrupting reconsolidation of fear memory in humans by a noradrenergic β-blocker.Cortical GluN2B deletion attenuates punished suppression of food reward-seeking.Reduction in Responding for Sucrose and Cocaine Reinforcement by Disruption of Memory Reconsolidation(1,2,3).Involvement of protein phosphatases in the destabilization of methamphetamine-associated contextual memoryAn appetitive experience after fear memory destabilization attenuates fear retention: involvement GluN2B-NMDA receptors in the Basolateral Amygdala Complex.The CB1 receptor antagonist AM251 impairs reconsolidation of pavlovian fear memory in the rat basolateral amygdala.Retrieval is not necessary to trigger reconsolidation of object recognition memory in the perirhinal cortex.Memory reconsolidation may be disrupted by a distractor stimulus presented during reactivation.Infralimbic GluN2A-Containing NMDA Receptors Modulate Reconsolidation of Cocaine Self-Administration Memory.Cued memory reconsolidation in rats requires nitric oxide.A Prediction Error-driven Retrieval Procedure for Destabilizing and Rewriting Maladaptive Reward Memories in Hazardous Drinkers.Nitric Oxide Upregulates Proteasomal Protein Degradation in Neurons.Pharmacologically induced amnesia for learned fear is time and sleep dependent.Dissociated Role of D-Serine in Extinction During Consolidation vs. Reconsolidation of Context Conditioned Fear.
P2860
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P2860
Double dissociation of the requirement for GluN2B- and GluN2A-containing NMDA receptors in the destabilization and restabilization of a reconsolidating memory.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
Double dissociation of the req ...... n of a reconsolidating memory.
@en
type
label
Double dissociation of the req ...... n of a reconsolidating memory.
@en
prefLabel
Double dissociation of the req ...... n of a reconsolidating memory.
@en
P2860
P50
P1476
Double dissociation of the req ...... n of a reconsolidating memory.
@en
P2093
Ben L Gregory
Jessica K Dumbreck
P2860
P304
P356
10.1523/JNEUROSCI.3273-12.2013
P407
P577
2013-01-01T00:00:00Z