Mxi1-0, an alternatively transcribed Mxi1 isoform, is overexpressed in glioblastomas.
about
A review of the past, present, and future directions of neoplasiaComing of Age in the Life ofNeoplasia: Where We Have Been and Where We Are GoingMxi1 and mxi1-0 antagonize N-myc function and independently mediate apoptosis in neuroblastomaInduction of Mxi1-SR alpha by FOXO3a contributes to repression of Myc-dependent gene expressionMxi1-0 regulates the growth of human umbilical vein endothelial cells through extracellular signal-regulated kinase 1/2 (ERK1/2) and interleukin-8 (IL-8)-dependent pathways.N-Myc differentially regulates expression of MXI1 isoforms in neuroblastoma.Human aging in the post-GWAS era: further insights reveal potential regulatory variants.LXR-α selectively reprogrammes cancer cells to enter into apoptosis.Oesophageal adenocarcinoma is associated with a deregulation in the MYC/MAX/MAD network.Differential effects of Mxi1-SRalpha and Mxi1-SRbeta in Myc antagonism.
P2860
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P2860
Mxi1-0, an alternatively transcribed Mxi1 isoform, is overexpressed in glioblastomas.
description
2004 nî lūn-bûn
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2004年の論文
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2004年論文
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2004年論文
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2004年論文
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2004年論文
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2004年論文
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2004年论文
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2004年论文
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2004年论文
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name
Mxi1-0, an alternatively transcribed Mxi1 isoform, is overexpressed in glioblastomas.
@en
type
label
Mxi1-0, an alternatively transcribed Mxi1 isoform, is overexpressed in glioblastomas.
@en
prefLabel
Mxi1-0, an alternatively transcribed Mxi1 isoform, is overexpressed in glioblastomas.
@en
P2093
P2860
P356
P1433
P1476
Mxi1-0, an alternatively transcribed Mxi1 isoform, is overexpressed in glioblastomas
@en
P2093
Andrew S Youkilis
Christy A Petroff
Daniel S Wechsler
Datong Zheng
Judith L Gorelick
Lars D Engstrom
Linda Q Benson
Melissa R Coon
Samir M Hanash
Valerie Ackley
P2860
P304
P356
10.1593/NEO.04244
P50
P577
2004-09-01T00:00:00Z