OSM Enhances Angiogenesis and Improves Cardiac Function after Myocardial Infarction.

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Oncostatin M-Preconditioned Mesenchymal Stem Cells Alleviate Bleomycin-Induced Pulmonary Fibrosis Through Paracrine Effects of the Hepatocyte Growth Factor The AB loop and D-helix in binding site III of human Oncostatin M (OSM) are required for OSM receptor activation.

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OSM Enhances Angiogenesis and Improves Cardiac Function after Myocardial Infarction.

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2015 nî lūn-bûn

@nan

2015年の論文

@ja

2015年論文

@yue

2015年論文

@zh-hant

2015年論文

@zh-hk

2015年論文

@zh-mo

2015年論文

@zh-tw

2015年论文

@wuu

2015年论文

@zh

2015年论文

@zh-cn

name

OSM Enhances Angiogenesis and Improves Cardiac Function after Myocardial Infarction.

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type

Item

label

OSM Enhances Angiogenesis and Improves Cardiac Function after Myocardial Infarction.

@en

prefLabel

OSM Enhances Angiogenesis and Improves Cardiac Function after Myocardial Infarction.

@en

P1476

OSM Enhances Angiogenesis and Improves Cardiac Function after Myocardial Infarction.

@en

P2093

Di Zhu

http://www.w3.org/2001/XMLSchema#string

Dongdong Sun

http://www.w3.org/2001/XMLSchema#string

Liping Wei

http://www.w3.org/2001/XMLSchema#string

Xiaotian Zhang

http://www.w3.org/2001/XMLSchema#string

Xin Qi

http://www.w3.org/2001/XMLSchema#string

Zhijing Zhao

http://www.w3.org/2001/XMLSchema#string

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Heart Disease and Stroke Statistics--2013 Update: A Report From the American Heart Association

5-Methoxyleoligin, a lignan from Edelweiss, stimulates CYP26B1-dependent angiogenesis in vitro and induces arteriogenesis in infarcted rat hearts in vivo

Oncostatin M mediates STAT3-dependent intestinal epithelial restitution via increased cell proliferation, decreased apoptosis and upregulation of SERPIN family members

Heart disease and stroke statistics--2014 update: a report from the American Heart Association

Pulsed electromagnetic field improves cardiac function in response to myocardial infarction.

Oncostatin M promotes STAT3 activation, VEGF production, and invasion in osteosarcoma cell lines.

Glutaredoxin-1 overexpression enhances neovascularization and diminishes ventricular remodeling in chronic myocardial infarction.

Thioredoxin 1 enhances neovascularization and reduces ventricular remodeling during chronic myocardial infarction: a study using thioredoxin 1 transgenic mice

Vascular endothelial growth factor (VEGF)-A: role on cardiac angiogenesis following myocardial infarction.

Akt1-mediated skeletal muscle growth attenuates cardiac dysfunction and remodeling after experimental myocardial infarction.

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scholarly article

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10.1155/2015/317905

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English

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2015

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Zongjin Li

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2015-06-04T00:00:00Z

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OSM Enhances Angiogenesis and Improves Cardiac Function after Myocardial Infarction.

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P2860

P2860

OSM Enhances Angiogenesis and Improves Cardiac Function after Myocardial Infarction.

description

name

type

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prefLabel

P1433

P1476

P2093

P2860

P304

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P356

P407

P478

P50

P577

P5875

P698

P932

P356

P1433

P1476

P2093

P2860

P304

P31

P356

P407

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P50

P577

P5875

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P932