Evidence for a critical role of GABAergic transmission within the thalamus in the genesis and control of absence seizures in the rat.
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Absence seizures in C3H/HeJ and knockout mice caused by mutation of the AMPA receptor subunit Gria4Control of Absence Seizures by the Thalamic Feed-Forward Inhibition.Refractory idiopathic absence status epilepticus: A probable paradoxical effect of phenytoin and carbamazepine.Interactions between membrane conductances underlying thalamocortical slow-wave oscillationsNew antiepileptic drugs: from serendipity to rational discovery.Disinhibition-induced transitions between absence and tonic-clonic epileptic seizuresNeurochemical and behavioral features in genetic absence epilepsy and in acutely induced absence seizures.Stimulus-induced Epileptic Spike-Wave Discharges in Thalamocortical Model with Disinhibition.Vigabatrin-induced generalized epileptiform discharges in a patient with focal epilepsyThe role of the basal ganglia in the control of seizure.The GABA(A) Receptor: Subunit-Dependent Functions and Absence Seizures.Anti-absence activity of mGlu1 and mGlu5 receptor enhancers and their interaction with a GABA reuptake inhibitor: Effect of local infusions in the somatosensory cortex and thalamus.Comparison of numbers of interneurons in three thalamic nuclei of normal and epileptic ratsPotentiation of mGlu5 receptors with the novel enhancer, VU0360172, reduces spontaneous absence seizures in WAG/Rij ratsEffects of adenosine administration on spike-wave discharge frequency in genetically epileptic rats.Metabolic approach of absence seizures in a genetic model of absence epilepsy, the GAERS: study of the leucine-glutamate cycle.Anticonvulsant actions of lamotrigine on spontaneous thalamocortical rhythms.GABA(B) receptor gene expression in monkey thalamus.Stimulus-induced transitions between spike-wave discharges and spindles with the modulation of thalamic reticular nucleus.The pacemaker role of thalamic reticular nucleus in controlling spike-wave discharges and spindles.Relations between cortical and thalamic cellular activities during absence seizures in rats.Focal administration of neuropeptide Y into the S2 somatosensory cortex maximally suppresses absence seizures in a genetic rat model.Do the quantitative relationships of synaptic junctions and terminals in the thalamus of genetic absence epilepsy rats from Strasbourg (GAERS) differ from those in normal control Wistar rats.On the origin and suddenness of absences in genetic absence models.Comparing GABAergic cell populations in the thalamic reticular nucleus of normal and genetic absence epilepsy rats from Strasbourg (GAERS).Bicuculline-induced rhythmic EEG episodes: gender differences and the effects of ethosuximide and baclofen treatment.Benzodiazepine-GABAA Receptor Binding During Absence SeizuresBenzodiazepine-GABAA Receptors in Idiopathic Generalized Epilepsy Measured with [11C]Flumazenil and Positron Emission Tomography
P2860
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P2860
Evidence for a critical role of GABAergic transmission within the thalamus in the genesis and control of absence seizures in the rat.
description
1991 nî lūn-bûn
@nan
1991年の論文
@ja
1991年論文
@yue
1991年論文
@zh-hant
1991年論文
@zh-hk
1991年論文
@zh-mo
1991年論文
@zh-tw
1991年论文
@wuu
1991年论文
@zh
1991年论文
@zh-cn
name
Evidence for a critical role o ...... f absence seizures in the rat.
@en
type
label
Evidence for a critical role o ...... f absence seizures in the rat.
@en
prefLabel
Evidence for a critical role o ...... f absence seizures in the rat.
@en
P2093
P1433
P1476
Evidence for a critical role o ...... f absence seizures in the rat.
@en
P2093
P356
10.1016/0006-8993(91)91262-Y
P577
1991-04-01T00:00:00Z