CXCR2 is required for neutrophilic airway inflammation and hyperresponsiveness in a mouse model of human rhinovirus infection. - Wikidata - awgldk - TriplyDB

CXCR2 is required for neutrophilic airway inflammation and hyperresponsiveness in a mouse model of human rhinovirus infection.

CXCR2 is required for neutrophilic airway inflammation and hyperresponsiveness in a mouse model of human rhinovirus infection.

http://www.wikidata.org/entity/Q41845119

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Effects of an anti-inflammatory VAP-1/SSAO inhibitor, PXS-4728A, on pulmonary neutrophil migration Genetic Ablation of CXCR2 Protects against Cigarette Smoke-Induced Lung Inflammation and Injury Altered Innate Immune Responses in Neutrophils from Patients with Well- and Suboptimally Controlled Asthma Cxcl8 (IL-8) mediates neutrophil recruitment and behavior in the zebrafish inflammatory response.Airway inflammation and illness severity in response to experimental rhinovirus infection in asthma.Macrophage activation state determines the response to rhinovirus infection in a mouse model of allergic asthma.Type I interferon signaling regulates Ly6C(hi) monocytes and neutrophils during acute viral pneumonia in mice MDA5 and TLR3 initiate pro-inflammatory signaling pathways leading to rhinovirus-induced airways inflammation and hyperresponsiveness.CXCR2 promotes ovarian cancer growth through dysregulated cell cycle, diminished apoptosis, and enhanced angiogenesis Lower respiratory tract infection induced by a genetically modified picornavirus in its natural murine host.Interleukin-12p40 modulates human metapneumovirus-induced pulmonary disease in an acute mouse model of infection PROPHYLACTIC ANTIBODY TREATMENT AND INTRAMUSCULAR IMMUNIZATION REDUCE INFECTIOUS HUMAN RHINOVIRUS 16 LOAD IN THE LOWER RESPIRATORY TRACT OF CHALLENGED COTTON RATS.Inhaled dsRNA and rhinovirus evoke neutrophilic exacerbation and lung expression of thymic stromal lymphopoietin in allergic mice with established experimental asthma The airway epithelium: soldier in the fight against respiratory viruses Intranasal administration of recombinant TRAIL down-regulates CXCL-1/KC in an ovalbumin-induced airway inflammation murine model Chemokine receptor CXCR2 mediates bacterial clearance rather than neutrophil recruitment in a murine model of pneumonic plague Rhinovirus exacerbates house-dust-mite induced lung disease in adult mice Neonatal hyperoxia contributes additively to cigarette smoke-induced chronic obstructive pulmonary disease changes in adult mice.CXC chemokines and antimicrobial peptides in rhinovirus-induced experimental asthma exacerbations.MicroRNA-26a/-26b-COX-2-MIP-2 Loop Regulates Allergic Inflammation and Allergic Inflammation-promoted Enhanced Tumorigenic and Metastatic Potential of Cancer Cells.Elastase/LPS-exposed mice exhibit impaired innate immune responses to bacterial challenge: role of scavenger receptor A.Coordinate activation of inflammatory gene networks, alveolar destruction and neonatal death in AKNA deficient mice.Chemokine CXCL1-Mediated Neutrophil Trafficking in the Lung: Role of CXCR2 Activation.Lung epithelial cells have virus-specific and shared gene expression responses to infection by diverse respiratory viruses.Neutrophil in viral infections, friend or foe?Double-stranded RNA induces molecular and inflammatory signatures that are directly relevant to COPD.Exposure to neonatal cigarette smoke causes durable lung changes but does not potentiate cigarette smoke-induced chronic obstructive pulmonary disease in adult mice.Bacterial Dose-Dependent Role of G Protein-Coupled Receptor Kinase 5 in Escherichia coli-Induced Pneumonia A novel immunomodulatory function of neutrophils on rhinovirus-activated monocytes in vitro Cytokine regulation of lung Th17 response to airway immunization using LPS adjuvant.Airway smooth muscle and immunomodulation in acute exacerbations of airway disease.Targeting airway smooth muscle in airways diseases: an old concept with new twists Rhinovirus-Induced Exacerbations of Asthma and COPD.Role of the CXCL8-CXCR1/2 Axis in Cancer and Inflammatory Diseases.Quercetin inhibits rhinovirus replication in vitro and in vivo.Rhinovirus Infection of ORMDL3 Transgenic Mice Is Associated with Reduced Rhinovirus Viral Load and Airway Inflammation.TRAIL signaling is proinflammatory and proviral in a murine model of rhinovirus 1B infection.TLR2 Activation Limits Rhinovirus-Stimulated CXCL-10 by Attenuating IRAK-1-Dependent IL-33 Receptor Signaling in Human Bronchial Epithelial Cells.Early life rhinovirus infection exacerbates house-dust-mite induced lung disease more severely in female mice.Rhinovirus infection interferes with induction of tolerance to aeroantigens through OX40 ligand, thymic stromal lymphopoietin, and IL-33.

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P2860

CXCR2 is required for neutrophilic airway inflammation and hyperresponsiveness in a mouse model of human rhinovirus infection.

http://www.wikidata.org/entity/Q41845119

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CXCR2 is required for neutroph ...... of human rhinovirus infection.

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CXCR2 is required for neutroph ...... of human rhinovirus infection.

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CXCR2 is required for neutroph ...... of human rhinovirus infection.

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CXCR2 is required for neutroph ...... of human rhinovirus infection.

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Deepti R Nagarkar

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Jee Shim

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Marc B Hershenson

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Nicholas W Lukacs

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Qiong Wang

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Uma Sajjan

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Wan C Tsai

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Ying Zhao

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P2860

Induction of functional IL-8 receptors by IL-4 and IL-13 in human monocytes

The major human rhinovirus receptor is ICAM-1

Rhinovirus infection induces cytotoxicity and delays wound healing in bronchial epithelial cells.

Expression and functional analysis of chemokine receptors in human peripheral blood leukocyte populations

A diverse group of previously unrecognized human rhinoviruses are common causes of respiratory illnesses in infants.

The neutrophil as a cellular source of chemokines.

Rhinovirus induces airway epithelial gene expression through double-stranded RNA and IFN-dependent pathways

Rhinovirus upper respiratory infection increases airway hyperreactivity and late asthmatic reactions.

Members of the low density lipoprotein receptor family mediate cell entry of a minor-group common cold virus

Infection of a human respiratory epithelial cell line with rhinovirus. Induction of cytokine release and modulation of susceptibility to infection by cytokine exposure.

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