Increased catalase expression improves muscle function in mdx mice
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Oxidative Stress-Mediated Skeletal Muscle Degeneration: Molecules, Mechanisms, and TherapiesRescue of dystrophic skeletal muscle by PGC-1α involves a fast to slow fiber type shift in the mdx mouseThe role of oxidative stress in skeletal muscle injury and regeneration: focus on antioxidant enzymesLong-Term Quercetin Dietary Enrichment Partially Protects Dystrophic Skeletal MuscleThe burden, epidemiology, costs and treatment for Duchenne muscular dystrophy: an evidence reviewA proteasome inhibitor fails to attenuate dystrophic pathology in mdx mice.Diapocynin, a dimer of the NADPH oxidase inhibitor apocynin, reduces ROS production and prevents force loss in eccentrically contracting dystrophic musclePorcine models of muscular dystrophyAbsence of Dystrophin Disrupts Skeletal Muscle Signaling: Roles of Ca2+, Reactive Oxygen Species, and Nitric Oxide in the Development of Muscular Dystrophy.Long-term wheel running compromises diaphragm function but improves cardiac and plantarflexor function in the mdx mouseContribution of oxidative stress to pathology in diaphragm and limb muscles with Duchenne muscular dystrophy.Oxidative stress and pathology in muscular dystrophies: focus on protein thiol oxidation and dysferlinopathies.α-Syntrophin stabilizes catalase to reduce endogenous reactive oxygen species levels during myoblast differentiation.Control of DNA integrity in skeletal muscle under physiological and pathological conditions.Oral quercetin administration transiently protects respiratory function in dystrophin-deficient mice.Improvement of endurance of DMD animal model using natural polyphenols.PGC-1α gene transfer improves muscle function in dystrophic muscle following prolonged disease progress.Utrophin influences mitochondrial pathology and oxidative stress in dystrophic muscleProteomic assessment of the acute phase of dystrophin deficiency in mdx mice.Proteomic profiling of cardiomyopathic tissue from the aged mdx model of Duchenne muscular dystrophy reveals a drastic decrease in laminin, nidogen and annexin.Rescue of dystrophic skeletal muscle by PGC-1α involves restored expression of dystrophin-associated protein complex components and satellite cell signaling.Autophagic dysfunction and autophagosome escape in the mdx mus musculus model of Duchenne muscular dystrophy.Oxidative stress in Duchenne muscular dystrophy: focus on the NRF2 redox pathway.
P2860
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P2860
Increased catalase expression improves muscle function in mdx mice
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
2010年论文
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2010年论文
@zh-cn
name
Increased catalase expression improves muscle function in mdx mice
@en
type
label
Increased catalase expression improves muscle function in mdx mice
@en
prefLabel
Increased catalase expression improves muscle function in mdx mice
@en
P2860
P1476
Increased catalase expression improves muscle function in mdx mice
@en
P2093
Joshua T Selsby
P2860
P304
P356
10.1113/EXPPHYSIOL.2010.054379
P577
2010-11-01T00:00:00Z