In situ expression of regulatory cytokines by heart inflammatory cells in Chagas' disease patients with heart failure. - Wikidata - awgldk - TriplyDB

In situ expression of regulatory cytokines by heart inflammatory cells in Chagas' disease patients with heart failure.

In situ expression of regulatory cytokines by heart inflammatory cells in Chagas' disease patients with heart failure.

http://www.wikidata.org/entity/Q41849739

about

The CXCL10/CXCR3 Axis and Cardiac Inflammation: Implications for Immunotherapy to Treat Infectious and Noninfectious Diseases of the Heart Omega-3 supplementation on inflammatory markers in patients with chronic Chagas cardiomyopathy: a randomized clinical study.Induction of IL-12 production in human peripheral monocytes by Trypanosoma cruzi Is mediated by glycosylphosphatidylinositol-anchored mucin-like glycoproteins and potentiated by IFN- γ and CD40-CD40L interactions.Tumor necrosis factor is a therapeutic target for immunological unbalance and cardiac abnormalities in chronic experimental Chagas' heart disease.Myocardial gene expression of T-bet, GATA-3, Ror-γt, FoxP3, and hallmark cytokines in chronic Chagas disease cardiomyopathy: an essentially unopposed TH1-type response.Presence of antigen-experienced T cells with low grade of differentiation and proliferative potential in chronic Chagas disease myocarditis.Chagas disease cardiomyopathy: immunopathology and genetics.Effects of omega-3 polyunsaturated fatty acid supplementation in patients with chronic chagasic cardiomyopathy: study protocol for a randomized controlled trial.Genetic susceptibility to Chagas disease cardiomyopathy: involvement of several genes of the innate immunity and chemokine-dependent migration pathways.Inflammation Enhances the Risks of Stroke and Death in Chronic Chagas Disease Patients.The heart of the matter: protection of the myocardium from T cells Could interferon-gamma be a therapeutic target for treating heart failure?The NO/ONOO-cycle as the central cause of heart failure.Chagas disease: still many unsolved issues.Evolution of anti-Trypanosoma cruzi antibody production in patients with chronic Chagas disease: Correlation between antibody titers and development of cardiac disease severity.Lack of Galectin-3 Prevents Cardiac Fibrosis and Effective Immune Responses in a Murine Model of Trypanosoma cruzi Infection.Depletion of regulatory T cells decreases cardiac parasitosis and inflammation in experimental Chagas disease.T cells responding to Trypanosoma cruzi detected by membrane TNF-α and CD154 in chagasic patients.Genetic susceptibility to Chagas disease: an overview about the infection and about the association between disease and the immune response genes.TGF-β Polymorphisms Are a Risk Factor for Chagas Disease.Correlation of transforming growth factor-β1 and tumour necrosis factor levels with left ventricular function in Chagas disease.CD86 Expression by Monocytes Influences an Immunomodulatory Profile in Asymptomatic Patients with Chronic Chagas Disease.

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P2860

In situ expression of regulatory cytokines by heart inflammatory cells in Chagas' disease patients with heart failure.

http://www.wikidata.org/entity/Q41849739

description

2012 nî lūn-bûn

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2012年の論文

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2012年論文

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2012年論文

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2012年论文

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2012年论文

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2012年论文

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name

In situ expression of regulato ...... e patients with heart failure.

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In situ expression of regulato ...... e patients with heart failure.

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In situ expression of regulato ...... e patients with heart failure.

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Clinical and Developmental Immunology

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In situ expression of regulato ...... e patients with heart failure.

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Audrey Romano

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Denise Bertulucci Rocha Rodrigues

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Marlene Antonia dos Reis

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Sanívia Aparecida de Lima Pereira

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Sebastião Tostes

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Vicente de Paula Antunes Teixeira

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Virmondes Rodrigues

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P2860

Immunological and clinical evaluation of chagasic patients subjected to chemotherapy during the acute phase of Trypanosoma cruzi infection 14-30 years ago

Genetic and functional role of TNF-alpha in the development Trypanosoma cruzi infection.

Clinical and epidemiological aspects of Chagas disease.

Nitric oxide is involved in control of Trypanosoma cruzi-induced parasitemia and directly kills the parasite in vitro.

Cardiac gene expression profiling provides evidence for cytokinopathy as a molecular mechanism in Chagas' disease cardiomyopathy.

Tumor necrosis factor alpha mediates resistance to Trypanosoma cruzi infection in mice by inducing nitric oxide production in infected gamma interferon-activated macrophages

Modulation of chemokine production and inflammatory responses in interferon-gamma- and tumor necrosis factor-R1-deficient mice during Trypanosoma cruzi infection.

The use of the Picrosirius-polarization method for the study of the biopathology of collagen.

In liver fibrosis, dendritic cells govern hepatic inflammation in mice via TNF-alpha.

Interferon-gamma-induced nitric oxide causes intrinsic intestinal denervation in Trypanosoma cruzi-infected mice.

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10.1155/2012/361730

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2012

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2012-07-03T00:00:00Z

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