In situ expression of regulatory cytokines by heart inflammatory cells in Chagas' disease patients with heart failure.
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The CXCL10/CXCR3 Axis and Cardiac Inflammation: Implications for Immunotherapy to Treat Infectious and Noninfectious Diseases of the HeartOmega-3 supplementation on inflammatory markers in patients with chronic Chagas cardiomyopathy: a randomized clinical study.Induction of IL-12 production in human peripheral monocytes by Trypanosoma cruzi Is mediated by glycosylphosphatidylinositol-anchored mucin-like glycoproteins and potentiated by IFN- γ and CD40-CD40L interactions.Tumor necrosis factor is a therapeutic target for immunological unbalance and cardiac abnormalities in chronic experimental Chagas' heart disease.Myocardial gene expression of T-bet, GATA-3, Ror-γt, FoxP3, and hallmark cytokines in chronic Chagas disease cardiomyopathy: an essentially unopposed TH1-type response.Presence of antigen-experienced T cells with low grade of differentiation and proliferative potential in chronic Chagas disease myocarditis.Chagas disease cardiomyopathy: immunopathology and genetics.Effects of omega-3 polyunsaturated fatty acid supplementation in patients with chronic chagasic cardiomyopathy: study protocol for a randomized controlled trial.Genetic susceptibility to Chagas disease cardiomyopathy: involvement of several genes of the innate immunity and chemokine-dependent migration pathways.Inflammation Enhances the Risks of Stroke and Death in Chronic Chagas Disease Patients.The heart of the matter: protection of the myocardium from T cellsCould interferon-gamma be a therapeutic target for treating heart failure?The NO/ONOO-cycle as the central cause of heart failure.Chagas disease: still many unsolved issues.Evolution of anti-Trypanosoma cruzi antibody production in patients with chronic Chagas disease: Correlation between antibody titers and development of cardiac disease severity.Lack of Galectin-3 Prevents Cardiac Fibrosis and Effective Immune Responses in a Murine Model of Trypanosoma cruzi Infection.Depletion of regulatory T cells decreases cardiac parasitosis and inflammation in experimental Chagas disease.T cells responding to Trypanosoma cruzi detected by membrane TNF-α and CD154 in chagasic patients.Genetic susceptibility to Chagas disease: an overview about the infection and about the association between disease and the immune response genes.TGF-β Polymorphisms Are a Risk Factor for Chagas Disease.Correlation of transforming growth factor-β1 and tumour necrosis factor levels with left ventricular function in Chagas disease.CD86 Expression by Monocytes Influences an Immunomodulatory Profile in Asymptomatic Patients with Chronic Chagas Disease.
P2860
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P2860
In situ expression of regulatory cytokines by heart inflammatory cells in Chagas' disease patients with heart failure.
description
2012 nî lūn-bûn
@nan
2012年の論文
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2012年論文
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2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
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2012年论文
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2012年论文
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name
In situ expression of regulato ...... e patients with heart failure.
@en
type
label
In situ expression of regulato ...... e patients with heart failure.
@en
prefLabel
In situ expression of regulato ...... e patients with heart failure.
@en
P2093
P2860
P356
P1476
In situ expression of regulato ...... e patients with heart failure.
@en
P2093
Audrey Romano
Denise Bertulucci Rocha Rodrigues
Marlene Antonia dos Reis
Sanívia Aparecida de Lima Pereira
Sebastião Tostes
Vicente de Paula Antunes Teixeira
Virmondes Rodrigues
P2860
P304
P356
10.1155/2012/361730
P577
2012-07-03T00:00:00Z