Rho-kinase signalling regulates trypsinogen activation and tissue damage in severe acute pancreatitis.
about
Review of experimental animal models of biliary acute pancreatitis and recent advances in basic researchPathogenic mechanisms of pancreatitis.Molecular mechanisms of pancreatic injury.Pathogenic mechanisms of acute pancreatitis.Endoplasmic reticulum stress is activated in acute pancreatitis.Emodin Alleviates Sodium Taurocholate-Induced Pancreatic Acinar Cell Injury via MicroRNA-30a-5p-Mediated Inhibition of High-Temperature Requirement A/Transforming Growth Factor Beta 1 Inflammatory Signaling.Inhibition of geranylgeranyltransferase attenuates neutrophil accumulation and tissue injury in severe acute pancreatitis.Streptococcal M1 protein-provoked CXC chemokine formation, neutrophil recruitment and lung damage are regulated by Rho-kinase signaling.Neutrophil-derived matrix metalloproteinase-9 is a potent activator of trypsinogen in acinar cells in acute pancreatitisRole of neutrophils in the activation of trypsinogen in severe acute pancreatitis
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P2860
Rho-kinase signalling regulates trypsinogen activation and tissue damage in severe acute pancreatitis.
description
2011 nî lūn-bûn
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2011年の論文
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2011年論文
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2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
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2011年論文
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2011年论文
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2011年论文
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2011年论文
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name
Rho-kinase signalling regulate ...... in severe acute pancreatitis.
@en
type
label
Rho-kinase signalling regulate ...... in severe acute pancreatitis.
@en
prefLabel
Rho-kinase signalling regulate ...... in severe acute pancreatitis.
@en
P2093
P2860
P1476
Rho-kinase signalling regulate ...... in severe acute pancreatitis.
@en
P2093
P2860
P304
P356
10.1111/J.1476-5381.2010.01060.X
P407
P577
2011-02-01T00:00:00Z