Repeated social defeat activates dendritic cells and enhances Toll-like receptor dependent cytokine secretion.
about
Neuroimmune mechanisms of stress: sex differences, developmental plasticity, and implications for pharmacotherapy of stress-related diseaseToll-like receptor mRNA expression is selectively increased in the colonic mucosa of two animal models relevant to irritable bowel syndromeStress-related programming of autonomic imbalance: role in allergy and asthma.Social stress enhances IL-1beta and TNF-alpha production by Porphyromonas gingivalis lipopolysaccharide-stimulated CD11b+ cells.Neural mechanisms linking social status and inflammatory responses to social stress.Laboratory environment and bio-medical experience: the impact of administration technique on the quality of immune-behavior data results in stress experienceAssessment of plasma C-reactive protein as a biomarker of posttraumatic stress disorder risk.Perinatal stress and early life programming of lung structure and functionPrenatal maternal stress and cord blood innate and adaptive cytokine responses in an inner-city cohort.Epinephrine-primed murine bone marrow-derived dendritic cells facilitate production of IL-17A and IL-4 but not IFN-γ by CD4+ T cells.Toll-like receptor 4: innate immune regulator of neuroimmune and neuroendocrine interactions in stress and major depressive disorderAssociation between plasma IL-6 response to acute stress and early-life adversity in healthy adults.Exposure to a social stressor alters the structure of the intestinal microbiota: implications for stressor-induced immunomodulation.Influenza virus-specific immunological memory is enhanced by repeated social defeat.The social environment and IL-6 in rats and humans.TLR4/NF-κB signaling contributes to chronic unpredictable mild stress-induced atherosclerosis in ApoE-/- mice.Social disruption induced priming of CNS inflammatory response to Theiler's virus is dependent upon stress induced IL-6 releaseStress and the anti-influenza immune response: repeated social defeat augments clonal expansion of CD8(+)T cells during primary influenza A viral infection.Enhanced peripheral toll-like receptor responses in psychosis: further evidence of a pro-inflammatory phenotype.Stressor-induced alterations of adaptive immunity to vaccination and viral pathogens.Peripheral and central effects of repeated social defeat stress: monocyte trafficking, microglial activation, and anxiety.Peripheral innate immune challenge exaggerated microglia activation, increased the number of inflammatory CNS macrophages, and prolonged social withdrawal in socially defeated mice.Induction of Suppressor Cells and Increased Tumor Growth following Chronic Psychosocial Stress in Male Mice.β-Adrenergic receptor mediated increases in activation and function of natural killer cells following repeated social disruptionBeta adrenergic blockade decreases the immunomodulatory effects of social disruption stress.Vagal nerve stimulation blocks interleukin 6-dependent synaptic hyperexcitability induced by lipopolysaccharide-induced acute stress in the rodent prefrontal cortex.Antagonistic pleiotropy at the human IL6 promoter confers genetic resilience to the pro-inflammatory effects of adverse social conditions in adolescence.Social stress up-regulates inflammatory gene expression in the leukocyte transcriptome via β-adrenergic induction of myelopoiesis.Social stress and asthma: the role of corticosteroid insensitivity.Non-nutrient causes of low-grade, systemic inflammation: support for a 'canary in the mineshaft' view of obesity in chronic disease.Neuroimmunology of the atherosclerotic plaque: a morphological approach.β-Adrenergic receptor antagonism prevents anxiety-like behavior and microglial reactivity induced by repeated social defeatSpleen contributes to restraint stress induced changes in blood leukocytes distributionRepeated Social Defeat, Neuroinflammation, and Behavior: Monocytes Carry the Signal.Neuroendocrine-immune circuits, phenotypes, and interactions.GABAergic modulation with classical benzodiazepines prevent stress-induced neuro-immune dysregulation and behavioral alterations.Immunogenic dendritic cells primed by social defeat enhance adaptive immunity to influenza A virus.Social disruption induces lung inflammation.Glucocorticoids mediate stress-induced priming of microglial pro-inflammatory responses.Behavior and pro-inflammatory cytokine variations among submissive and dominant mice engaged in aggressive encounters: moderation by corticosterone reactivity.
P2860
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P2860
Repeated social defeat activates dendritic cells and enhances Toll-like receptor dependent cytokine secretion.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Repeated social defeat activat ...... dependent cytokine secretion.
@en
type
label
Repeated social defeat activat ...... dependent cytokine secretion.
@en
prefLabel
Repeated social defeat activat ...... dependent cytokine secretion.
@en
P2093
P2860
P1476
Repeated social defeat activat ...... dependent cytokine secretion.
@en
P2093
D A Padgett
L M Stiner-Jones
Nicole D Powell
P2860
P304
P356
10.1016/J.BBI.2008.09.010
P577
2008-09-26T00:00:00Z