Inhaled nitric oxide reduces secondary brain damage after traumatic brain injury in mice. - Wikidata - awgldk - TriplyDB

Inhaled nitric oxide reduces secondary brain damage after traumatic brain injury in mice.

Inhaled nitric oxide reduces secondary brain damage after traumatic brain injury in mice.

http://www.wikidata.org/entity/Q41870259

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Emerging therapies in traumatic brain injury Traumatic brain injury disrupts cerebrovascular tone through endothelial inducible nitric oxide synthase expression and nitric oxide gain of function Phosphodiesterase Inhibitors as a Therapeutic Approach to Neuroprotection and Repair.Chronic cerebrovascular dysfunction after traumatic brain injury Far-red tracer analysis of traumatic cerebrovascular permeability.Endothelial nitric oxide synthase mediates the cerebrovascular effects of erythropoietin in traumatic brain injury Augmenting collateral blood flow during ischemic stroke via transient aortic occlusion.Fresh frozen plasma resuscitation provides neuroprotection compared to normal saline in a large animal model of traumatic brain injury and polytrauma.Decompressive craniectomy reduces white matter injury after controlled cortical impact in mice.Inhaled Nitric Oxide as an Adjunctive Treatment for Cerebral Malaria in Children: A Phase II Randomized Open-Label Clinical Trial.Brief inhalation of nitric oxide increases resuscitation success and improves 7-day-survival after cardiac arrest in rats: a randomized controlled animal study.Molecular contributions to neurovascular unit dysfunctions after brain injuries: lessons for target-specific drug development.The role of inhaled nitric oxide beyond ARDS.Cerebral collaterals and collateral therapeutics for acute ischemic stroke.The Role of Nitric Oxide and Sympathetic Control in Cerebral Autoregulation in the Setting of Subarachnoid Hemorrhage and Traumatic Brain Injury.Gas Diffusion in the CNS.Apolipoprotein E Mimetic Peptide Increases Cerebral Glucose Uptake by Reducing Blood-Brain Barrier Disruption after Controlled Cortical Impact in Mice: An 18F-Fluorodeoxyglucose PET/CT Study.Nitric oxide inhalation reduces brain damage, prevents mortality, and improves neurological outcome after subarachnoid hemorrhage by resolving early pial microvasospasms.Cylindromatosis mediates neuronal cell death in vitro and in vivo.Impact of inhaled nitric oxide on white matter damage in growth-restricted neonatal rats.Hydrogen sulfide, nitric oxide, and neurodegenerative disorders.Central Infusion of IGF-1 Increases Hippocampal Neurogenesis and Improves Neurobehavioral Function following Traumatic Brain Injury.The Role of Gaseous Molecules in Traumatic Brain Injury: An Updated Review.

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Inhaled nitric oxide reduces secondary brain damage after traumatic brain injury in mice.

http://www.wikidata.org/entity/Q41870259

description

2012 nî lūn-bûn

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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2012年论文

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2012年论文

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name

Inhaled nitric oxide reduces secondary brain damage after traumatic brain injury in mice.

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type

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Inhaled nitric oxide reduces secondary brain damage after traumatic brain injury in mice.

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Inhaled nitric oxide reduces secondary brain damage after traumatic brain injury in mice.

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Journal of Cerebral Blood Flow & Metabolism

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Inhaled nitric oxide reduces secondary brain damage after traumatic brain injury in mice.

@en

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Nicole A Terpolilli

http://www.w3.org/2001/XMLSchema#string

Nikolaus Plesnila

http://www.w3.org/2001/XMLSchema#string

Seong-Woong Kim

http://www.w3.org/2001/XMLSchema#string

Serge C Thal

http://www.w3.org/2001/XMLSchema#string

Wolfgang M Kuebler

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P2860

FELASA guidelines and recommendations

Effects of inhaled nitric oxide on regional blood flow are consistent with intravascular nitric oxide delivery

Improving bioscience research reporting: the ARRIVE guidelines for reporting animal research

Cerebral hemodynamic effects of phenylephrine and L-arginine after cortical impact injury.

Edema and brain trauma.

The effects of L-arginine on cerebral hemodynamics after controlled cortical impact injury in the mouse.

L-arginine and free radical scavengers increase cerebral blood flow and brain tissue nitric oxide concentrations after controlled cortical impact injury in rats.

The role of endothelial nitric oxide synthase in the cerebral hemodynamics after controlled cortical impact injury in mice.

Changes of cerebral blood flow during the secondary expansion of a cortical contusion assessed by 14C-iodoantipyrine autoradiography in mice using a non-invasive protocol.

BID mediates neuronal cell death after oxygen/ glucose deprivation and focal cerebral ischemia

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311-318

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scholarly article

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10.1038/JCBFM.2012.176

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2

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33

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2012-11-28T00:00:00Z

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233789174

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23188422

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traumatic brain injury

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3564204

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