Caudal regression in adrenocortical dysplasia (acd) mice is caused by telomere dysfunction with subsequent p53-dependent apoptosis.
about
Zebrafish as a model system to study the physiological function of telomeric protein TPP1TPP1 is required for TERT recruitment, telomere elongation during nuclear reprogramming, and normal skin development in miceMBTPS1/SKI-1/S1P proprotein convertase is required for ECM signaling and axial elongation during somitogenesis and vertebral development†.Additive effect of TAp63 deficiency on the adrenocortical dysplasia (acd) phenotype.Sacral agenesis: a pilot whole exome sequencing and copy number study.The shelterin complex and hematopoiesis.Transcriptome profiling reveals differentially expressed transcripts between the human adrenal zona fasciculata and zona reticularis.Telomeres and telomerase in cancerHematopoietic stem cells are acutely sensitive to Acd shelterin gene inactivation.Defending the end zone: studying the players involved in protecting chromosome endsAdrenocortical growth and cancer.Control of Cellular Aging, Tissue Function, and Cancer by p53 Downstream of Telomeres.
P2860
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P2860
Caudal regression in adrenocortical dysplasia (acd) mice is caused by telomere dysfunction with subsequent p53-dependent apoptosis.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
2009年论文
@zh
2009年论文
@zh-cn
name
Caudal regression in adrenocor ...... quent p53-dependent apoptosis.
@en
type
label
Caudal regression in adrenocor ...... quent p53-dependent apoptosis.
@en
prefLabel
Caudal regression in adrenocor ...... quent p53-dependent apoptosis.
@en
P2093
P2860
P1476
Caudal regression in adrenocor ...... quent p53-dependent apoptosis.
@en
P2093
Andrea S Krause
Bridget C O'Connor
Catherine E Keegan
Christopher N Vlangos
Gail A Osawa
Madeleine J Morley
P2860
P304
P356
10.1016/J.YDBIO.2009.07.038
P407
P577
2009-08-03T00:00:00Z