Increased expression of histone deacetylaces (HDACs) and inhibition of prostate cancer growth and invasion by HDAC inhibitor SAHA.
about
A transcriptional repressor co-regulatory network governing androgen response in prostate cancersChemoprevention of mouse lung and colon tumors by suberoylanilide hydroxamic acid and atorvastatin.Chemopreventive mechanisms of α-keto acid metabolites of naturally occurring organoselenium compounds.miRNA-34b inhibits prostate cancer through demethylation, active chromatin modifications, and AKT pathways.The anti-tumor effects and molecular mechanisms of suberoylanilide hydroxamic acid (SAHA) on the aggressive phenotypes of ovarian carcinoma cellsIdentification of novel microRNA regulatory pathways associated with heterogeneous prostate cancer.The synthesis and evaluation of N1-(4-(2-[18F]-fluoroethyl)phenyl)-N8-hydroxyoctanediamide ([18F]-FESAHA), a PET radiotracer designed for the delineation of histone deacetylase expression in cancer.A new method for discovering disease-specific MiRNA-target regulatory networksRegulators of gene expression as biomarkers for prostate cancerCombination therapy: histone deacetylase inhibitors and platinum-based chemotherapeutics for cancerInvestigation of the expression patterns and correlation of DNA methyltransferases and class I histone deacetylases in ovarian cancer tissues.Histone deacetylases and mechanisms of regulation of gene expressionThe in vitro and vivo anti-tumor effects and molecular mechanisms of suberoylanilide hydroxamic acid (SAHA) and MG132 on the aggressive phenotypes of gastric cancer cells.A novel method for isolation of histones from serum and its implications in therapeutics and prognosis of solid tumoursThe role of REST and HDAC2 in epigenetic dysregulation of Nav1.5 and nNav1.5 expression in breast cancer.Intensified antineoplastic effect by combining an HDAC-inhibitor, an mTOR-inhibitor and low dosed interferon alpha in prostate cancer cells.Histone deacetylase activity mediates acquired resistance towards structurally diverse HSP90 inhibitors.Green tea polyphenols causes cell cycle arrest and apoptosis in prostate cancer cells by suppressing class I histone deacetylasesEngineering Lineage Potency and Plasticity of Stem Cells using Epigenetic Molecules
P2860
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P2860
Increased expression of histone deacetylaces (HDACs) and inhibition of prostate cancer growth and invasion by HDAC inhibitor SAHA.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
2009年论文
@zh
2009年论文
@zh-cn
name
Increased expression of histon ...... vasion by HDAC inhibitor SAHA.
@en
type
label
Increased expression of histon ...... vasion by HDAC inhibitor SAHA.
@en
prefLabel
Increased expression of histon ...... vasion by HDAC inhibitor SAHA.
@en
P2093
P2860
P1476
Increased expression of histon ...... nvasion by HDAC inhibitor SAHA
@en
P2093
Aaron D Berger
Andrew Wilton
Christian Twiss
Hongfeng Guo
Iman Osman
Jason Chiu
Longgui Wang
William Gerald
Xuanyi Zou
P2860
P407
P577
2009-01-01T00:00:00Z