12-Chemokine gene signature identifies lymph node-like structures in melanoma: potential for patient selection for immunotherapy? - Wikidata - awgldk - TriplyDB

12-Chemokine gene signature identifies lymph node-like structures in melanoma: potential for patient selection for immunotherapy?

12-Chemokine gene signature identifies lymph node-like structures in melanoma: potential for patient selection for immunotherapy?

http://www.wikidata.org/entity/Q41883987

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Tertiary Lymphoid Organs in Cancer Tissues Predictive factors for immunotherapy in melanoma The Next Hurdle in Cancer Immunotherapy: Overcoming the Non-T-Cell-Inflamed Tumor Microenvironment Reflections on the Histopathology of Tumor-Infiltrating Lymphocytes in Melanoma and the Host Immune Response Control of CD8 T-Cell Infiltration into Tumors by Vasculature and Microenvironment Therapeutic Lymphoid Organogenesis in the Tumor Microenvironment Pathways and therapeutic targets in melanoma Micrometastatic cancer cells in lymph nodes, bone marrow, and blood: Clinical significance and biologic implications Innate and adaptive immune cells in the tumor microenvironment Characteristics of tertiary lymphoid structures in primary cancers Tertiary Lymphoid Structures in Cancers: Prognostic Value, Regulation, and Manipulation for Therapeutic Intervention Molecular pathways: inflammation-associated nitric-oxide production as a cancer-supporting redox mechanism and a potential therapeutic target Heterogeneity of CD8+ tumor-infiltrating lymphocytes in non-small-cell lung cancer: impact on patient prognostic assessments and comparison of quantification by different sampling strategies Topical treatment of melanoma metastases with imiquimod, plus administration of a cancer vaccine, promotes immune signatures in the metastases.Effector lymphocyte-induced lymph node-like vasculature enables naive T-cell entry into tumours and enhanced anti-tumour immunity Chemokine receptor patterns in lymphocytes mirror metastatic spreading in melanoma.A case of spontaneous systemic immunity to melanoma associated with cure after amputation for extensive regional recurrence.Peripheral tissue homing receptor control of naïve, effector, and memory CD8 T cell localization in lymphoid and non-lymphoid tissues.Direct T cell-tumour interaction triggers TH1 phenotype activation through the modification of the mesenchymal stromal cells transcriptional programme.Evaluation of invasive breast cancer samples using a 12-chemokine gene expression score: correlation with clinical outcomes Immunotherapy converts nonimmunogenic pancreatic tumors into immunogenic foci of immune regulation Intramuscular therapeutic vaccination targeting HPV16 induces T cell responses that localize in mucosal lesions.Ectopic lymphoid structures function as microniches for tumor progenitor cells in hepatocellular carcinoma.Activated lymphocyte recruitment into the tumor microenvironment following preoperative sipuleucel-T for localized prostate cancer Tertiary Lymphoid Structure-Associated B Cells are Key Players in Anti-Tumor Immunity.Tertiary lymphoid structures are associated with higher tumor grade in primary operable breast cancer patients.Decoding the regulatory landscape of melanoma reveals TEADS as regulators of the invasive cell state.Antitumor effector B cells directly kill tumor cells via the Fas/FasL pathway and are regulated by IL-10 Prognostic implications of PD-L1 expression in patients with soft tissue sarcoma.Image Analysis of the Tumor Microenvironment.Safety, correlative markers, and clinical results of adjuvant nivolumab in combination with vaccine in resected high-risk metastatic melanoma.Spatial distribution of B cells predicts prognosis in human pancreatic adenocarcinoma.Long-lasting complete regression of established mouse tumors by counteracting Th2 inflammation Extranodal induction of therapeutic immunity in the tumor microenvironment after intratumoral delivery of Tbet gene-modified dendritic cells CXCR3/CCR5 pathways in metastatic melanoma patients treated with adoptive therapy and interleukin-2.Defective immunogenic cell death of HMGB1-deficient tumors: compensatory therapy with TLR4 agonists.A novel non-Hodgkin lymphoma murine model closer to the standard clinical scenario.An independent validation of a gene expression signature to differentiate malignant melanoma from benign melanocytic nevi.Therapeutic use of dendritic cells to promote the extranodal priming of anti-tumor immunity.Genetic alterations and personalized medicine in melanoma: progress and future prospects.

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12-Chemokine gene signature identifies lymph node-like structures in melanoma: potential for patient selection for immunotherapy?

http://www.wikidata.org/entity/Q41883987

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12-Chemokine gene signature id ...... t selection for immunotherapy?

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Scientific Reports

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12-Chemokine gene signature id ...... t selection for immunotherapy?

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Anders E Berglund

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David A Fenstermacher

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James J Mulé

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Jane L Messina

http://www.w3.org/2001/XMLSchema#string

Jeffrey S Weber

http://www.w3.org/2001/XMLSchema#string

Mark C Lloyd

http://www.w3.org/2001/XMLSchema#string

Michael J Schell

http://www.w3.org/2001/XMLSchema#string

Steven Eschrich

http://www.w3.org/2001/XMLSchema#string

Vernon K Sondak

http://www.w3.org/2001/XMLSchema#string

Xiaotao Qu

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Durable complete responses in heavily pretreated patients with metastatic melanoma using T-cell transfer immunotherapy

The blockade of immune checkpoints in cancer immunotherapy

Cancer and the chemokine network

Immune checkpoint proteins: a new therapeutic paradigm for cancer--preclinical background: CTLA-4 and PD-1 blockade

CCR6 regulates the migration of inflammatory and regulatory T cells

On the eve of personalized medicine in oncology

The many roles of chemokines and chemokine receptors in inflammation

Analysis of T-cell clonality using laser capture microdissection and high-resolution microcapillary electrophoresis.

Ectopic B-cell clusters that infiltrate transplanted human kidneys are clonal

Two hundred years of cancer research.

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765

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10.1038/SREP00765

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1

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2

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2012-10-24T00:00:00Z

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232700723

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23097687

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3479449

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