UmuD and RecA directly modulate the mutagenic potential of the Y family DNA polymerase DinB.
about
Eukaryotic translesion polymerases and their roles and regulation in DNA damage toleranceCulture history and population heterogeneity as determinants of bacterial adaptation: the adaptomics of a single environmental transitionThree Residues of the Interdomain Linker Determine the Conformation and Single-base Deletion Fidelity of Y-family Translesion PolymerasesSeparation of recombination and SOS response in Escherichia coli RecA suggests LexA interaction sitesAn active site aromatic triad in Escherichia coli DNA Pol IV coordinates cell survival and mutagenesis in different DNA damaging agentsA Genetic Selection for dinB Mutants Reveals an Interaction between DNA Polymerase IV and the Replicative Polymerase That Is Required for Translesion SynthesisConformational dynamics of the Escherichia coli DNA polymerase manager proteins UmuD and UmuD'.UmuD(2) inhibits a non-covalent step during DinB-mediated template slippage on homopolymeric nucleotide runs.Whither the replisome: emerging perspectives on the dynamic nature of the DNA replication machinery.Genetic requirement for mutagenesis of the G[8,5-Me]T cross-link in Escherichia coli: DNA polymerases IV and V compete for error-prone bypass.The unusual UBZ domain of Saccharomyces cerevisiae polymerase ηThe SOS Regulatory Network.Translesion DNA polymerasesThe SMC-like protein complex SbcCD enhances DNA polymerase IV-dependent spontaneous mutation in Escherichia coliTranslesion DNA Synthesis.The dimeric SOS mutagenesis protein UmuD is active as a monomerEscherichia coli Rep DNA helicase and error-prone DNA polymerase IV interact physically and functionally(5'S)-8,5'-cyclo-2'-deoxyguanosine is a strong block to replication, a potent pol V-dependent mutagenic lesion, and is inefficiently repaired in Escherichia coliRecombinase and translesion DNA polymerase decrease the speed of replication fork progression during the DNA damage response in Escherichia coli cells.Efficient extension of slipped DNA intermediates by DinB is required to escape primer template realignment by DnaQ.Escherichia coli nucleoside diphosphate kinase mutants depend on translesion DNA synthesis to prevent mutagenesis.Interactions and Localization of Escherichia coli Error-Prone DNA Polymerase IV after DNA Damage.Diverse responses to UV light exposure in Acinetobacter include the capacity for DNA damage-induced mutagenesis in the opportunistic pathogens Acinetobacter baumannii and Acinetobacter ursingiiSelective disruption of the DNA polymerase III α-β complex by the umuD gene products.Altering the N-terminal arms of the polymerase manager protein UmuD modulates protein interactionsRegulation of Escherichia coli SOS mutagenesis by dimeric intrinsically disordered umuD gene products.Antibiotic resistance acquired through a DNA damage-inducible response in Acinetobacter baumannii.A single residue unique to DinB-like proteins limits formation of the polymerase IV multiprotein complex in Escherichia coli.Low-fidelity DNA synthesis by human DNA polymerase theta.RecA acts as a switch to regulate polymerase occupancy in a moving replication fork.Mutations for Worse or Better: Low-Fidelity DNA Synthesis by SOS DNA Polymerase V Is a Tightly Regulated Double-Edged SwordPreferential D-loop extension by a translesion DNA polymerase underlies error-prone recombination.Translesion DNA polymerases remodel the replisome and alter the speed of the replicative helicase.DinB upregulation is the sole role of the SOS response in stress-induced mutagenesis in Escherichia coli.Polymerase manager protein UmuD directly regulates Escherichia coli DNA polymerase III α binding to ssDNA.A global view of antibiotic resistance.Characterization of novel alleles of the Escherichia coli umuDC genes identifies additional interaction sites of UmuC with the beta clamp.DNA polymerases are error-prone at RecA-mediated recombination intermediates.A DinB variant reveals diverse physiological consequences of incomplete TLS extension by a Y-family DNA polymerase.Multiple strategies for translesion synthesis in bacteria
P2860
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P2860
UmuD and RecA directly modulate the mutagenic potential of the Y family DNA polymerase DinB.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
UmuD and RecA directly modulat ...... Y family DNA polymerase DinB.
@en
type
label
UmuD and RecA directly modulat ...... Y family DNA polymerase DinB.
@en
prefLabel
UmuD and RecA directly modulat ...... Y family DNA polymerase DinB.
@en
P2093
P2860
P1433
P1476
UmuD and RecA directly modulat ...... Y family DNA polymerase DinB.
@en
P2093
Graham C Walker
Sharotka M Simon
Valentin Ilyin
Veronica G Godoy
P2860
P304
P356
10.1016/J.MOLCEL.2007.10.025
P577
2007-12-01T00:00:00Z