Glucocorticoid protects rodent hearts from ischemia/reperfusion injury by activating lipocalin-type prostaglandin D synthase-derived PGD2 biosynthesis.
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Enzymes of the cyclooxygenase pathways of prostanoid biosynthesisThe contribution of prostaglandins versus prostacyclin in ventricular remodeling during heart failureStructural Basis of the Catalytic Mechanism Operating in Open-Closed Conformers of Lipocalin Type Prostaglandin D SynthaseDual role for glucocorticoids in cardiomyocyte hypertrophy and apoptosisRole of G3BP1 in glucocorticoid receptor-mediated microRNA-15b and microRNA-23a biogenesis in endothelial cells.Mitochondrial dysfunction and reduced prostaglandin synthesis in skeletal muscle of Group VIB Ca2+-independent phospholipase A2gamma-deficient mice.Construction of microRNA and transcription factor regulatory network based on gene expression data in cardiomyopathyInduction of Galphas contributes to the paradoxical stimulation of cytosolic phospholipase A2alpha expression by cortisol in human amnion fibroblasts.Glucocorticoid modulates angiotensin II receptor expression patterns and protects the heart from ischemia and reperfusion injury.FoxO transcription factors promote cardiomyocyte survival upon induction of oxidative stress.Dexamethasone protects neonatal hypoxic-ischemic brain injury via L-PGDS-dependent PGD2-DP1-pERK signaling pathway.High-Quality Protein Crystal Growth of Mouse Lipocalin-Type Prostaglandin D Synthase in Microgravity.Lipocalin-type prostaglandin D2 synthase protein regulates glial cell migration and morphology through myristoylated alanine-rich C-kinase substrate: prostaglandin D2-independent effectsOxygen surrounding the heart during ischemic conservation determines the myocardial injury during reperfusionPGD synthase and PGD2 in immune resposne.Deleterious effect of the IL-23/IL-17A axis and γδT cells on left ventricular remodeling after myocardial infarction.Antenatal hypoxia induces epigenetic repression of glucocorticoid receptor and promotes ischemic-sensitive phenotype in the developing heart.An eicosanoid-centric view of atherothrombotic risk factorsEssential role of stress hormone signaling in cardiomyocytes for the prevention of heart disease.Tissue-specific actions of glucocorticoids on apoptosis: a double-edged sword.Transcriptome remodeling in hypoxic inflammation.Glucocorticoids promote Von Hippel Lindau degradation and Hif-1α stabilization.Non-linear actions of physiological agents: Finite disarrangements elicit fitness benefits.TIPE2 acts as a negative regulator linking NOD2 and inflammatory responses in myocardial ischemia/reperfusion injury.15-deoxy-Δ¹²,¹⁴-PGJ₂ promotes inflammation and apoptosis in cardiomyocytes via the DP2/MAPK/TNFα axis.Investigations of Glucocorticoid Action in GN.Dexamethasone conjugation to polyamidoamine dendrimers G1 and G2 for enhanced transfection efficiency with an anti-inflammatory effect.Krüppel-like Factor 13 Is a Major Mediator of Glucocorticoid Receptor Signaling in Cardiomyocytes and Protects These Cells from DNA Damage and Death.Intrathecal corticoids in permanent focal cerebral ischemia in rats. Part I: a new therapeutic approach in the acute phase.Hypothalamic-Pituitary-Adrenal Axis Modulation of Glucocorticoids in the Cardiovascular System.MicroRNA-210 suppresses glucocorticoid receptor expression in response to hypoxia in fetal rat cardiomyocytes.Budesonide instillation immediately after reperfusion ameliorates ischemia/reperfusion-induced injury in the transplanted lung of rat.Niacin Promotes Cardiac Healing after Myocardial Infarction through Activation of the Myeloid Prostaglandin D2 Receptor Subtype 1.CRTH2 promotes endoplasmic reticulum stress-induced cardiomyocyte apoptosis through m-calpain.L-PGDS Mediates Vagus Nerve Stimulation-Induced Neuroprotection in a Rat Model of Ischemic Stroke by Suppressing the Apoptotic Response.Identification of potentially relevant genes for myocardial infarction using RNA sequencing data analysis.Adenosine Production by Biomaterial-Supported Mesenchymal Stromal Cells Reduces the Innate Inflammatory Response in Myocardial Ischemia/Reperfusion Injury.Correlation of serum- and glucocorticoid-regulated kinase 1 expression with ischemia-reperfusion injury after heart transplantation.Nrf2 Deficiency Unmasks the Significance of Nitric Oxide Synthase Activity for Cardioprotection.
P2860
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P2860
Glucocorticoid protects rodent hearts from ischemia/reperfusion injury by activating lipocalin-type prostaglandin D synthase-derived PGD2 biosynthesis.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
2009年论文
@zh
2009年论文
@zh-cn
name
Glucocorticoid protects rodent ...... ase-derived PGD2 biosynthesis.
@en
type
label
Glucocorticoid protects rodent ...... ase-derived PGD2 biosynthesis.
@en
prefLabel
Glucocorticoid protects rodent ...... ase-derived PGD2 biosynthesis.
@en
P2093
P2860
P356
P1476
Glucocorticoid protects rodent ...... ase-derived PGD2 biosynthesis.
@en
P2093
Hidenori Moriyama
Hiroki Nakanishi
Hirotoshi Tanaka
Kayoko Tamaki
Keiichi Fukuda
Ken Shinmura
Kengo Tomita
Koichiro Asano
Mitsushige Murata
P2860
P304
P356
10.1172/JCI37413
P407
P577
2009-05-18T00:00:00Z