Tumor necrosis factor alpha-induced activation of c-jun N-terminal kinase is mediated by TRAF2.
about
TRAF family proteins interact with the common neurotrophin receptor and modulate apoptosis inductionCleavage of the death domain kinase RIP by caspase-8 prompts TNF-induced apoptosisCIKS, a connection to Ikappa B kinase and stress-activated protein kinaseThe tyrosine kinase Syk regulates TPL2 activation signalsTRANCE is a novel ligand of the tumor necrosis factor receptor family that activates c-Jun N-terminal kinase in T cellsHuman glutathione S-transferase P1-1 interacts with TRAF2 and regulates TRAF2-ASK1 signalsEpstein-Barr virus-encoded latent membrane protein 1 activates the JNK pathway through its extreme C terminus via a mechanism involving TRADD and TRAF2Ankyrin repeat and SOCS box 3 (ASB3) mediates ubiquitination and degradation of tumor necrosis factor receptor IIJAMP, a Jun N-terminal kinase 1 (JNK1)-associated membrane protein, regulates duration of JNK activity.Signaling by proinflammatory cytokines: oligomerization of TRAF2 and TRAF6 is sufficient for JNK and IKK activation and target gene induction via an amino-terminal effector domainTumor necrosis factor (TNF)-mediated kinase cascades: bifurcation of nuclear factor-kappaB and c-jun N-terminal kinase (JNK/SAPK) pathways at TNF receptor-associated factor 2The human toll signaling pathway: divergence of nuclear factor kappaB and JNK/SAPK activation upstream of tumor necrosis factor receptor-associated factor 6 (TRAF6)CD28-independent, TRAF2-dependent costimulation of resting T cells by 4-1BB ligandA splicing variant of a death domain protein that is regulated by a mitogen-activated kinase is a substrate for c-Jun N-terminal kinase in the human central nervous systemThe zinc finger protein A20 inhibits TNF-induced NF-kappaB-dependent gene expression by interfering with an RIP- or TRAF2-mediated transactivation signal and directly binds to a novel NF-kappaB-inhibiting protein ABINRIP2 is a Raf1-activated mitogen-activated protein kinase kinaseDisruption of hsp90 function results in degradation of the death domain kinase, receptor-interacting protein (RIP), and blockage of tumor necrosis factor-induced nuclear factor-kappaB activationNF-kappaB activation through IKK-i-dependent I-TRAF/TANK phosphorylationPleiotropic signal transduction mediated by human CD30: a member of the tumor necrosis factor receptor (TNFR) familyCharacterization of the intracellular domain of receptor activator of NF-kappaB (RANK). Interaction with tumor necrosis factor receptor-associated factors and activation of NF-kappab and c-Jun N-terminal kinaseTumour necrosis factor receptor 1 mediates endoplasmic reticulum stress-induced activation of the MAP kinase JNKNuclear factor kB-independent cytoprotective pathways originating at tumor necrosis factor receptor-associated factor 2CD40 signaling of monocyte inflammatory cytokine synthesis through an ERK1/2-dependent pathway. A target of interleukin (il)-4 and il-10 anti-inflammatory actionIdentification of Grb2 as a novel binding partner of tumor necrosis factor (TNF) receptor I.Estrogen decreases osteoclast formation by down-regulating receptor activator of NF-kappa B ligand (RANKL)-induced JNK activation.Signaling events following chemokine receptor ligation in human dendritic cells at different developmental stages.On the role of tumor necrosis factor and receptors in models of multiorgan failure, rheumatoid arthritis, multiple sclerosis and inflammatory bowel disease.Hepatitis C virus core protein enhances NF-kappaB signal pathway triggering by lymphotoxin-beta receptor ligand and tumor necrosis factor alpha.Mechanisms of apoptosis by c-Myc.Activation of hPAK65 by caspase cleavage induces some of the morphological and biochemical changes of apoptosis.Role of the TRAF binding site and NF-kappaB activation in Epstein-Barr virus latent membrane protein 1-induced cell gene expressionMatrix metalloproteinase 9 expression is induced by Epstein-Barr virus latent membrane protein 1 C-terminal activation regions 1 and 2.The death domain kinase RIP is essential for TRAIL (Apo2L)-induced activation of IkappaB kinase and c-Jun N-terminal kinase.Role of TNF in lymphocyte-mediated cytotoxicity.Jun N-terminal kinase 2 modulates thymocyte apoptosis and T cell activation through c-Jun and nuclear factor of activated T cell (NF-AT)The role of the death-domain kinase RIP in tumour-necrosis-factor-induced activation of mitogen-activated protein kinasesTL1, a novel tumor necrosis factor-like cytokine, induces apoptosis in endothelial cells. Involvement of activation of stress protein kinases (stress-activated protein kinase and p38 mitogen-activated protein kinase) and caspase-3-like protease.Glucocorticoid-independent repression of tumor necrosis factor (TNF) alpha-stimulated interleukin (IL)-6 expression by the glucocorticoid receptor: a potential mechanism for protection against an excessive inflammatory response.Thalidomide Analogues Suppress Lipopolysaccharide-Induced Synthesis of TNF-α and Nitrite, an Intermediate of Nitric Oxide, in a Cellular Model of InflammationTRAF2 exerts its antiapoptotic effect by regulating the expression of Krüppel-like factor LKLF
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P2860
Tumor necrosis factor alpha-induced activation of c-jun N-terminal kinase is mediated by TRAF2.
description
1997 nî lūn-bûn
@nan
1997年の論文
@ja
1997年論文
@yue
1997年論文
@zh-hant
1997年論文
@zh-hk
1997年論文
@zh-mo
1997年論文
@zh-tw
1997年论文
@wuu
1997年论文
@zh
1997年论文
@zh-cn
name
Tumor necrosis factor alpha-in ...... l kinase is mediated by TRAF2.
@en
type
label
Tumor necrosis factor alpha-in ...... l kinase is mediated by TRAF2.
@en
prefLabel
Tumor necrosis factor alpha-in ...... l kinase is mediated by TRAF2.
@en
P2093
P2860
P356
P1433
P1476
Tumor necrosis factor alpha-in ...... l kinase is mediated by TRAF2.
@en
P2093
Reinhard C
Shyamala V
Williams LT
P2860
P304
P356
10.1093/EMBOJ/16.5.1080
P407
P577
1997-03-01T00:00:00Z