Oxidation of a potassium channel causes progressive sensory function loss during aging.
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Oxidation of K(+) Channels in Aging and NeurodegenerationIntracellular redox state alters NMDA receptor response during aging through Ca2+/calmodulin-dependent protein kinase IIThe lack of CuZnSOD leads to impaired neurotransmitter release, neuromuscular junction destabilization and reduced muscle strength in mice.Oxidative modulation of voltage-gated potassium channels.Phospholipase A2 - nexus of aging, oxidative stress, neuronal excitability, and functional decline of the aging nervous system? Insights from a snail model system of neuronal aging and age-associated memory impairmentGenetic analysis of age-dependent defects of the Caenorhabditis elegans touch receptor neuronsA common copy number variation (CNV) polymorphism in the CNTNAP4 gene: association with aging in femalesGuanine nucleotide exchange factor OSG-1 confers functional aging via dysregulated Rho signaling in Caenorhabditis elegans neuronsDecreased response to acetylcholine during aging of aplysia neuron R15.Single-cell redox imaging demonstrates a distinctive response of dopaminergic neurons to oxidative insultsKChIP-like auxiliary subunits of Kv4 channels regulate excitability of muscle cells and control male turning behavior during mating in Caenorhabditis elegansKCNE Regulation of K(+) Channel Trafficking - a Sisyphean Task?Molecular mechanisms underlying the apoptotic effect of KCNB1 K+ channel oxidation.A Caenorhabditis elegans model system for amylopathy studyOxidation of KCNB1 Potassium Channels Causes Neurotoxicity and Cognitive Impairment in a Mouse Model of Traumatic Brain Injury.Neuronal aging: learning from C. elegansChemistry and the worm: Caenorhabditis elegans as a platform for integrating chemical and biological research.Redox Signaling in Neurotransmission and Cognition During Aging.SIR-2.1 integrates metabolic homeostasis with the reproductive neuromuscular excitability in early aging male Caenorhabditis elegans.Neuroprotective effects of Hu-Yi-Neng, a diet supplement, on SH-SY5Y human neuroblastoma cells.Membrane ion Channels and Receptors in Animal lifespan Modulation.Redox-sensitive sulfenic acid modification regulates surface expression of the cardiovascular voltage-gated potassium channel Kv1.5.Cysteines control the N- and C-linker-dependent gating of KCNH1 potassium channels.The neurotoxic effects of hydrogen peroxide and copper in Retzius nerve cells of the leech Haemopis sanguisuga.Consumption of fig fruits grown in Oman can improve memory, anxiety, and learning skills in a transgenic mice model of Alzheimer's disease.Oxidation of KCNB1 channels in the human brain and in mouse model of Alzheimer's disease
P2860
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P2860
Oxidation of a potassium channel causes progressive sensory function loss during aging.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
2009年论文
@zh
2009年论文
@zh-cn
name
Oxidation of a potassium channel causes progressive sensory function loss during aging.
@en
type
label
Oxidation of a potassium channel causes progressive sensory function loss during aging.
@en
prefLabel
Oxidation of a potassium channel causes progressive sensory function loss during aging.
@en
P2860
P356
P1433
P1476
Oxidation of a potassium channel causes progressive sensory function loss during aging.
@en
P2093
Federico Sesti
Shi-Qing Cai
P2860
P2888
P304
P356
10.1038/NN.2291
P407
P577
2009-03-29T00:00:00Z
P5875
P6179
1016148620