ATM deficiency augments constitutively nuclear cyclin D1-driven genomic instability and lymphomagenesis.
about
The potential for chemical mixtures from the environment to enable the cancer hallmark of sustained proliferative signallingDNA damage signaling guards against perturbation of cyclin D1 expression triggered by low-dose long-term fractionated radiationPERK Is a Haploinsufficient Tumor Suppressor: Gene Dose Determines Tumor-Suppressive Versus Tumor Promoting Properties of PERK in Melanoma.Cyclin D1, cancer progression, and opportunities in cancer treatment.FBXO4 loss facilitates carcinogen induced papilloma development in mice.A lowered 26S proteasome activity correlates with mantle lymphoma cell lines resistance to genotoxic stress.Early B-cell-specific inactivation of ATM synergizes with ectopic CyclinD1 expression to promote pre-germinal center B-cell lymphomas in mice.PRMT5 is required for lymphomagenesis triggered by multiple oncogenic drivers
P2860
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P2860
ATM deficiency augments constitutively nuclear cyclin D1-driven genomic instability and lymphomagenesis.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
ATM deficiency augments consti ...... stability and lymphomagenesis.
@en
type
label
ATM deficiency augments consti ...... stability and lymphomagenesis.
@en
prefLabel
ATM deficiency augments consti ...... stability and lymphomagenesis.
@en
P2093
P2860
P356
P1433
P1476
ATM deficiency augments consti ...... stability and lymphomagenesis.
@en
P2093
C H Bassing
L P Vaites
P2860
P2888
P304
P356
10.1038/ONC.2012.577
P407
P577
2013-01-14T00:00:00Z
P5875
P6179
1052276345