An oxidized extracellular oxidation-reduction state increases Nox1 expression and proliferation in vascular smooth muscle cells via epidermal growth factor receptor activation.
about
Biochemistry, physiology, and pathophysiology of NADPH oxidases in the cardiovascular systemRedox signaling in cardiovascular pathophysiology: A focus on hydrogen peroxide and vascular smooth muscle cellsHu antigen R is required for NOX-1 but not NOX-4 regulation by inflammatory stimuli in vascular smooth muscle cells.Impact of exudative diathesis induced by selenium deficiency on LncRNAs and their roles in the oxidative reduction process in broiler chick veins.Nox4 NADPH oxidase contributes to smooth muscle cell phenotypes associated with unstable atherosclerotic plaquesCysteine/cystine redox signaling in cardiovascular disease.Activation of NADPH oxidase 1 increases intracellular calcium and migration of smooth muscle cells.Protein disulfide isomerase expression increases in resistance arteries during hypertension development. Effects on Nox1 NADPH oxidase signaling.Xanthine oxidase-derived ROS upregulate Egr-1 via ERK1/2 in PA smooth muscle cells; model to test impact of extracellular ROS in chronic hypoxia.Nox1 transactivation of epidermal growth factor receptor promotes N-cadherin shedding and smooth muscle cell migrationRegulation of NAD(P)H oxidases by AMPK in cardiovascular systemsThe multifunctional Ca2+/calmodulin-dependent kinase II regulates vascular smooth muscle migration through matrix metalloproteinase 9.Increased epidermal growth factor-like ligands are associated with elevated vascular nicotinamide adenine dinucleotide phosphate oxidase in a primate model of atherosclerosis.The x-ray absorption spectroscopy model of solvation about sulfur in aqueous L-cysteineCullin-5, a ubiquitin ligase scaffold protein, is significantly underexpressed in endometrial adenocarcinomas and is a target of miR-182.Inhibition of glutathione biosynthesis alters compartmental redox status and the thiol proteome in organogenesis-stage rat conceptusesThe epidermal growth factor receptor and its ligands in cardiovascular disease.Nox1 NADPH oxidase is necessary for late but not early myocardial ischaemic preconditioning.The Nox family of NADPH oxidases: friend or foe of the vascular system?Differential roles of NADPH oxidases in vascular physiology and pathophysiologyPhysiological roles of NOX/NADPH oxidase, the superoxide-generating enzyme.Glutathione peroxidase-deficient smooth muscle cells cause paracrine activation of normal smooth muscle cells via cyclophilin A.NOX4-derived reactive oxygen species limit fibrosis and inhibit proliferation of vascular smooth muscle cells in diabetic atherosclerosis.Increased expression of Nox1 in neointimal smooth muscle cells promotes activation of matrix metalloproteinase-9.The NOX-ROS connection: targeting Nox1 control of N-cadherin shedding in vascular smooth muscle cells.Tuning of the pro-oxidant and antioxidant activity of trolox through the controlled release from biodegradable poly(trolox ester) polymers.
P2860
Q26991619-C807B368-F21D-4401-865C-C12E4071D908Q28071377-1860F602-0B1D-49BB-ABA1-8AA794776864Q30786020-FA417C9A-4D00-43B6-906D-DE5AEF47B4A7Q33591305-DAA92ECF-90E1-4EF3-90D8-3C0EE44669C7Q33737346-22132833-3A8B-404C-B57D-8E652709C0DCQ34580141-239243BB-CE8B-4587-AC6C-035C162F3E42Q35178441-9E20BB97-BEE1-452A-B019-4AD93C958BBCQ35222727-A28B2837-C08C-4683-A2CE-FB40433B66C6Q35576738-66CAF710-33F4-4310-B0A6-A6EA037561D7Q35766044-290DD0B5-E9BB-4EF5-BB77-66F06A711F4DQ35925959-DB6F9432-D092-4D13-914E-E777A4410561Q35994104-639D8418-9147-43AF-B09D-779C1EBAAF81Q36319377-17A3AD48-A9C3-42ED-A6A3-B3B2760925C8Q36477631-BDDCDAF1-8913-4B4D-8827-B03FE5FC494EQ36641178-25FA508F-CB78-4F72-A60E-3F060BABC45BQ37151275-A0151D5E-55B6-4CC4-8DF3-294F0280E529Q37291330-0BDE7634-FFC0-4C57-BC40-7F652C1F4B8DQ37644770-74F66846-922F-434C-B298-35CE44C83A75Q37954511-BCF93A3F-6042-4776-AA13-D1961D991175Q37971795-BC30E9EE-1ACE-40F3-8E34-6082E2D28A88Q37976066-5E213B45-CA35-40D1-9F30-FDE780B7D262Q39167570-D3138F31-A011-4200-977D-245CFFAFDB9BQ42160197-E14E62E4-0037-4E5D-B32B-22F5366E602FQ42186142-123A8BAC-88D2-4B3D-9026-9A881E576FFDQ42664082-4E1353DF-EF0E-43E0-9B95-39C60084DD63Q46068672-F9DCFB78-100C-4B92-8DDE-3FEA39A5AD2A
P2860
An oxidized extracellular oxidation-reduction state increases Nox1 expression and proliferation in vascular smooth muscle cells via epidermal growth factor receptor activation.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
2010年论文
@zh
2010年论文
@zh-cn
name
An oxidized extracellular oxid ...... th factor receptor activation.
@en
type
label
An oxidized extracellular oxid ...... th factor receptor activation.
@en
prefLabel
An oxidized extracellular oxid ...... th factor receptor activation.
@en
P2093
P2860
P1476
An oxidized extracellular oxid ...... th factor receptor activation.
@en
P2093
Bojana Stanic
Francis J Miller
Masato Katsuyama
P2860
P304
P356
10.1161/ATVBAHA.110.207639
P407
P577
2010-09-02T00:00:00Z