The viral ubiquitin ligase ICP0 is neither sufficient nor necessary for degradation of the cellular DNA sensor IFI16 during herpes simplex virus 1 infection.
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Role of ND10 nuclear bodies in the chromatin repression of HSV-1Dynamic Response of IFI16 and Promyelocytic Leukemia Nuclear Body Components to Herpes Simplex Virus 1 InfectionProteomic approaches to uncovering virus-host protein interactions during the progression of viral infectionThe Role of Nuclear Antiviral Factors against Invading DNA Viruses: The Immediate Fate of Incoming Viral GenomesEvasion of host antiviral innate immunity by HSV-1, an updateViral evasion of intracellular DNA and RNA sensingIFI16 restricts HSV-1 replication by accumulating on the hsv-1 genome, repressing HSV-1 gene expression, and directly or indirectly modulating histone modificationsInnate nuclear sensor IFI16 translocates into the cytoplasm during the early stage of in vitro human cytomegalovirus infection and is entrapped in the egressing virions during the late stage.HSV-1 ICP0: An E3 Ubiquitin Ligase That Counteracts Host Intrinsic and Innate ImmunityNovel roles of cytoplasmic ICP0: proteasome-independent functions of the RING finger are required to block interferon-stimulated gene production but not to promote viral replication.cGAS-mediated stabilization of IFI16 promotes innate signaling during herpes simplex virus infection.Nuclear sensing of viral DNA, epigenetic regulation of herpes simplex virus infection, and innate immunityAnalysis of the SUMO2 Proteome during HSV-1 Infection.Interactions of the Antiviral Factor Interferon Gamma-Inducible Protein 16 (IFI16) Mediate Immune Signaling and Herpes Simplex Virus-1 ImmunosuppressionThe emerging role of nuclear viral DNA sensors.A quantitative assay to monitor HSV-1 ICP0 ubiquitin ligase activity in vitroNuclear Innate Immune DNA Sensor IFI16 Is Degraded during Lytic Reactivation of Kaposi's Sarcoma-Associated Herpesvirus (KSHV): Role of IFI16 in Maintenance of KSHV Latency.Viral DNA Sensors IFI16 and Cyclic GMP-AMP Synthase Possess Distinct Functions in Regulating Viral Gene Expression, Immune Defenses, and Apoptotic Responses during Herpesvirus Infection.Post-translational regulation of inflammasomes.HSV-1 degrades, stabilizes, requires, or is stung by STING depending on ICP0, the US3 protein kinase, and cell derivation.IFI16 and cGAS cooperate in the activation of STING during DNA sensing in human keratinocytes.Cellular sensing of viral DNA and viral evasion mechanisms.A proteomics perspective on viral DNA sensors in host defense and viral immune evasion mechanisms.Activation and regulation of DNA-driven immune responses.Neurons versus herpes simplex virus: the innate immune interactions that contribute to a host-pathogen standoffEvasion of the STING DNA-Sensing Pathway by VP11/12 of Herpes Simplex Virus 1.Impaired STING Pathway in Human Osteosarcoma U2OS Cells Contributes to the Growth of ICP0-Null Mutant Herpes Simplex Virus.Stimulation of the Replication of ICP0-Null Mutant Herpes Simplex Virus 1 and pp71-Deficient Human Cytomegalovirus by Epstein-Barr Virus Tegument Protein BNRF1.Relative Contributions of Herpes Simplex Virus 1 ICP0 and vhs to Loss of Cellular IFI16 Vary in Different Human Cell Types.Novel Role for Protein Inhibitor of Activated STAT 4 (PIAS4) in the Restriction of Herpes Simplex Virus 1 by the Cellular Intrinsic Antiviral Immune Response.Cellular Protein WDR11 Interacts with Specific Herpes Simplex Virus Proteins at the trans-Golgi Network To Promote Virus Replication.Diverse mechanisms evolved by DNA viruses to inhibit early host defenses.Innate Immune Mechanisms and Herpes Simplex Virus Infection and Disease.Regulatory Interaction between the Cellular Restriction Factor IFI16 and Viral pp65 (pUL83) Modulates Viral Gene Expression and IFI16 Protein Stability.Human Antiviral Protein IFIX Suppresses Viral Gene Expression during Herpes Simplex Virus 1 (HSV-1) Infection and Is Counteracted by Virus-induced Proteasomal Degradation.MORC3, a Component of PML Nuclear Bodies, Has a Role in Restricting Herpes Simplex Virus 1 and Human Cytomegalovirus.HSV-1 ICP27 targets the TBK1-activated STING signalsome to inhibit virus-induced type I IFN expressionThe functional interactome of PYHIN immune regulators reveals IFIX is a sensor of viral DNA.Interferon-γ-inducible protein 16 (IFI16) is required for the maintenance of Epstein-Barr virus latency.Distinct temporal roles for the promyelocytic leukaemia (PML) protein in the sequential regulation of intracellular host immunity to HSV-1 infection.
P2860
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P2860
The viral ubiquitin ligase ICP0 is neither sufficient nor necessary for degradation of the cellular DNA sensor IFI16 during herpes simplex virus 1 infection.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
The viral ubiquitin ligase ICP ...... pes simplex virus 1 infection.
@en
type
label
The viral ubiquitin ligase ICP ...... pes simplex virus 1 infection.
@en
prefLabel
The viral ubiquitin ligase ICP ...... pes simplex virus 1 infection.
@en
P2093
P2860
P356
P1433
P1476
The viral ubiquitin ligase ICP ...... rpes simplex virus 1 infection
@en
P2093
Delphine Cuchet-Lourenço
Gail Anderson
Roger D Everett
P2860
P304
13422-13432
P356
10.1128/JVI.02474-13
P407
P577
2013-10-02T00:00:00Z