Aging causes decreased resistance to multiple stresses and a failure to activate specific stress response pathways - Wikidata - awgldk - TriplyDB

Aging causes decreased resistance to multiple stresses and a failure to activate specific stress response pathways

Aging causes decreased resistance to multiple stresses and a failure to activate specific stress response pathways

http://www.wikidata.org/entity/Q41894661

about

SKN-1-independent transcriptional activation of glutathione S-transferase 4 (GST-4) by EGF signaling.'Molecular habituation' as a potential mechanism of gradual homeostatic loss with age.Uncoupling of oxidative stress resistance and lifespan in long-lived isp-1 mitochondrial mutants in Caenorhabditis elegans.Measuring Oxidative Stress in Caenorhabditis elegans: Paraquat and Juglone Sensitivity Assays.Activation of the mitochondrial unfolded protein response promotes longevity and dopamine neuron survival in Parkinson's disease models.Differential effects of chronic stress in young-adult and old female mice: cognitive-behavioral manifestations and neurobiological correlates.DPFF-1 transcription factor deficiency causes the aberrant activation of MPK-1 and meiotic defects in the Caenorhabditis elegans germline.Mechanisms underlying longevity: A genetic switch model of aging.Antagonistic pleiotropy and mutation accumulation contribute to age-related decline in stress response.Activation of DAF-16/FOXO by reactive oxygen species contributes to longevity in long-lived mitochondrial mutants in Caenorhabditis elegans.Epigenetic Mechanisms Impacting Aging: A Focus on Histone Levels and Telomeres.

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P2860

Aging causes decreased resistance to multiple stresses and a failure to activate specific stress response pathways

http://www.wikidata.org/entity/Q41894661

description

2016 nî lūn-bûn

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2016年の論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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2016年论文

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2016年论文

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2016年论文

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name

Aging causes decreased resista ...... cific stress response pathways

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Aging causes decreased resista ...... cific stress response pathways

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Alexis L Bergsma

http://www.w3.org/2001/XMLSchema#string

Claire E Schaar

http://www.w3.org/2001/XMLSchema#string

Dylan J Dues

http://www.w3.org/2001/XMLSchema#string

Emily K Andrews

http://www.w3.org/2001/XMLSchema#string

Jeremy M Van Raamsdonk

http://www.w3.org/2001/XMLSchema#string

Megan M Senchuk

http://www.w3.org/2001/XMLSchema#string

P2860

Deletion of the mitochondrial superoxide dismutase sod-2 extends lifespan in Caenorhabditis elegans

Thermotolerance and extended life-span conferred by single-gene mutations and induced by thermal stress

Is the oxidative stress theory of aging dead?

The DAF-16 FOXO transcription factor regulates natc-1 to modulate stress resistance in Caenorhabditis elegans, linking insulin/IGF-1 signaling to protein N-terminal acetylation

A mitochondrial superoxide signal triggers increased longevity in Caenorhabditis elegans

Surveillance-Activated Defenses Block the ROS–Induced Mitochondrial Unfolded Protein Response

A decline in p38 MAPK signaling underlies immunosenescence in Caenorhabditis elegans

A HIF-independent mediator of transcriptional responses to oxygen deprivation in Caenorhabditis elegans.

Aging: a theory based on free radical and radiation chemistry

A C. elegans mutant that lives twice as long as wild type

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777-795

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scholarly article

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10.18632/AGING.100939

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4

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8

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4925828

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