Effect of cyclic AMP-dependent hormones and Ca2+-mobilizing hormones on the Ca2+ influx and polyphosphoinositide metabolism in isolated rat hepatocytes.
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Convergent and parallel activation of low-conductance potassium channels by calcium and cAMP-dependent protein kinase.The role of dietary nucleotides in single-stomached animals.Role of phosphoinositides in the regulation of liver function.Second messengers and the regulation of Ca2+ fluxes by Ca2+-mobilizing agonists in rat liver.Calcium: its modulation in liver by cross-talk between the actions of glucagon and calcium-mobilizing agonists.Effects of catecholamines on plasma potassium: the role of alpha- and beta-adrenoceptors.Effects of glucagon and Ca2+ on the metabolism of phosphatidylinositol 4-phosphate and phosphatidylinositol 4,5-bisphosphate in isolated rat hepatocytes and plasma membranes.Glucagon and vasopressin interactions on Ca2+ movements in isolated hepatocytes.Modulation of cytosolic-[Ca2+] oscillations in hepatocytes results from cross-talk among second messengers. The synergism between the alpha 1-adrenergic response, glucagon and cyclic AMP, and their antagonism by insulin and diacylglycerol manifest tAnti-diabetic biguanides inhibit hormone-induced intracellular Ca2+ concentration oscillations in rat hepatocytes.Stimulation of hepatic inositol 1,4,5-trisphosphate kinase activity by Ca2+-dependent and -independent mechanisms.Exposure of cultured hepatocytes to cyclic AMP enhances the vasopressin-mediated stimulation of inositol phosphate production.Multiple mechanisms by which protein kinase A potentiates inositol 1,4,5-trisphosphate-induced Ca2+ mobilization in permeabilized hepatocytes.Conditions that result in the mobilization and influx of Ca2+ into rat hepatocytes induce the rapid loss of 3-hydroxy-3-methylglutaryl-CoA reductase activity that is not reversed by phosphatase treatment.Hepatic inositol release upon hormonal stimulation of perfused rat liver.Different modulatory effects of elevated cyclic AMP on cytosolic Ca2+ spikes induced by phenylephrine or vasopressin in single rat hepatocytes.Frequency and amplitude enhancement of calcium transients by cyclic AMP in hepatocytes.How far does phospholipase C activity depend on the cell calcium concentration? A study in intact cells.Prostaglandin F2 alpha and the thromboxane A2 analogue ONO-11113 stimulate Ca2+ fluxes and other physiological responses in rat liver. Further evidence that prostanoids may be involved in the action of arachidonic acid and platelet-activating factorCyclic AMP-evoked oscillations of intracellular [Ca2+] in guinea-pig hepatocytes.Effects of angiotensin II and nonpeptide receptor antagonists on transduction pathways in rat proximal tubule.Effects of phorbol esters and forskolin on basal and histamine-induced accumulation of inositol phosphates in cultured bovine adrenal chromaffin cells.Inorganic pyrophosphate is located primarily in the mitochondria of the hepatocyte and increases in parallel with the decrease in light-scattering induced by gluconeogenic hormones, butyrate and ionophore A23187.Evidence that stimulation of plasma-membrane Ca2+ inflow is an early action of glucagon and dibutyryl cyclic AMP in rat hepatocytes.Characterization of Ca2+ fluxes in rat liver plasma-membrane vesiclesElevated intracellular cyclic AMP exerts different modulatory effects on cytosolic free Ca2+ oscillations induced by ADP and ATP in single rat hepatocytes.The influx of Ca2+ induced by the administration of glucagon and Ca2+-mobilizing agents to the perfused rat liver could involve at least two separate pathways.Liver mitochondrial pyrophosphate concentration is increased by Ca2+ and regulates the intramitochondrial volume and adenine nucleotide content.Phosphatidic acid and arachidonic acid each interact synergistically with glucagon to stimulate Ca2+ influx in the perfused rat liverRapid Ca2+ influx induced by the action of dibutylhydroquinone and glucagon in the perfused rat liver.Protein kinase A regulates the disposition of Ca2+ which enters the cytoplasmic space through store-activated Ca2+ channels in rat hepatocytes by diverting inflowing Ca2+ to mitochondria.Permissive role of cAMP in the oscillatory Ca2+ response to inositol 1,4,5-trisphosphate in rat hepatocytes.Evidence indicating that the glucagon-induced increase in cytoplasmic free Ca2+ concentration in hepatocytes is mediated by an increase in cyclic AMP concentration.Activation of inositol phosphate formation by circulating noradrenaline but not by sympathetic nerve stimulation with a similar increase of glucose release in perfused rat liver.Does the inositol tris/tetrakisphosphate pathway exist in rat heart?
P2860
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P2860
Effect of cyclic AMP-dependent hormones and Ca2+-mobilizing hormones on the Ca2+ influx and polyphosphoinositide metabolism in isolated rat hepatocytes.
description
1986 nî lūn-bûn
@nan
1986年の論文
@ja
1986年論文
@yue
1986年論文
@zh-hant
1986年論文
@zh-hk
1986年論文
@zh-mo
1986年論文
@zh-tw
1986年论文
@wuu
1986年论文
@zh
1986年论文
@zh-cn
name
Effect of cyclic AMP-dependent ...... m in isolated rat hepatocytes.
@en
type
label
Effect of cyclic AMP-dependent ...... m in isolated rat hepatocytes.
@en
prefLabel
Effect of cyclic AMP-dependent ...... m in isolated rat hepatocytes.
@en
P2093
P2860
P356
P1433
P1476
Effect of cyclic AMP-dependent ...... m in isolated rat hepatocytes.
@en
P2093
P2860
P304
P356
10.1042/BJ2350663
P407
P577
1986-05-01T00:00:00Z